A gene(s) within the H-2D region determines the development of toxoplasmic encephalitis in mice.
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Effects of Toxoplasma gondii infection on the brainSusceptibility to chronic infection with Toxoplasma gondii does not correlate with susceptibility to acute infection in mice.Cytokine mRNA in the central nervous system of SCID mice infected with Toxoplasma gondii: importance of T-cell-independent regulation of resistance to T. gondii.Impact of regulated secretion on antiparasitic CD8 T cell responses.Expression of Toxoplasma gondii-specific heat shock protein 70 during In vivo conversion of bradyzoites to tachyzoites.VCAM-1/α4β1 integrin interaction is crucial for prompt recruitment of immune T cells into the brain during the early stage of reactivation of chronic infection with Toxoplasma gondii to prevent toxoplasmic encephalitis.Toxoplasma gondii: 25 years and 25 major advances for the fieldRegulation and function of T-cell-mediated immunity during Toxoplasma gondii infection.DNA vaccination with genes encoding Toxoplasma gondii antigens GRA1, GRA7, and ROP2 induces partially protective immunity against lethal challenge in mice.Requirement of non-T cells that produce gamma interferon for prevention of reactivation of Toxoplasma gondii infection in the brainInnate refractoriness of the Lewis rat to toxoplasmosis is a dominant trait that is intrinsic to bone marrow-derived cellsProtective Toxoplasma gondii-specific T-cell responses require T-cell-specific expression of protein kinase C-theta.The Toxoplasma gondii peptide AS15 elicits CD4 T cells that can control parasite burden.Toxoplasma IgG and IgA, but not IgM, antibody titers increase in sera of immunocompetent mice in association with proliferation of tachyzoites in the brain during the chronic stage of infectionA GRA1 DNA vaccine primes cytolytic CD8(+) T cells to control acute Toxoplasma gondii infectionResistance to Toxoplasma gondii in mice infected as neonates or exposed in utero.CXCL9 is important for recruiting immune T cells into the brain and inducing an accumulation of the T cells to the areas of tachyzoite proliferation to prevent reactivation of chronic cerebral infection with Toxoplasma gondii.MyD88-dependent protective immunity elicited by adenovirus 5 expressing the surface antigen 1 from Toxoplasma gondii is mediated by CD8(+) T lymphocytes.Prevalence of infection with Toxoplasma gondii in landrace and mixed breed pigs slaughtered in Baja California Sur State, Mexico.Different roles for interleukin-4 during the course of Toxoplasma gondii infection.Recombinant GRA4 or ROP2 protein combined with alum or the gra4 gene provides partial protection in chronic murine models of toxoplasmosisImpaired resistance to the development of toxoplasmic encephalitis in interleukin-6-deficient mice.Toxoplasma encephalitis in congenic B10 and BALB mice: impact of genetic factors on the immune response.Toxoplasma gondii antigens recognized by IgG antibodies differ between mice with and without active proliferation of tachyzoites in the brain during the chronic stage of infection.Presentation of Toxoplasma gondii antigens via the endogenous major histocompatibility complex class I pathway in nonprofessional and professional antigen-presenting cells.Role of cytokines and major histocompatibility complex restriction in mouse resistance to infection with a natural recombinant strain (type I-III) of Toxoplasma gondiiVaccination with Toxoplasma gondii SAG-1 protein is protective against congenital toxoplasmosis in BALB/c mice but not in CBA/J mice.Gamma interferon production, but not perforin-mediated cytolytic activity, of T cells is required for prevention of toxoplasmic encephalitis in BALB/c mice genetically resistant to the disease.Shared Immune and Repair Markers During Experimental Toxoplasma Chronic Brain Infection and SchizophreniaHeme oxygenase-1 activity is involved in the control of Toxoplasma gondii infection in the lung of BALB/c and C57BL/6 and in the small intestine of C57BL/6 mice.A toxoplasma patatin-like protein changes localization and alters the cytokine response during toxoplasmic encephalitis.Determination of genotypes of Toxoplasma gondii strains isolated from patients with toxoplasmosis.Determination of a Key Antigen for Immunological Intervention To Target the Latent Stage of Toxoplasma gondii.Involvement of Host Defense Mechanisms against Toxoplasma gondii Infection in Anhedonic and Despair-Like Behaviors in MiceTransnuclear CD8 T cells specific for the immunodominant epitope Gra6 lower acute-phase Toxoplasma gondii burdenT Cell Receptor-Major Histocompatibility Complex Interaction Strength Defines Trafficking and CD103+ Memory Status of CD8 T Cells in the Brain.BALB/c mice resistant to Toxoplasma gondii infection proved to be highly susceptible when previously infected with Myocoptes musculinus fur mitesImpact of Engineered Expression of Mitochondrial Association Factor 1b on Infection and the Host Response in a Mouse Model
P2860
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P2860
A gene(s) within the H-2D region determines the development of toxoplasmic encephalitis in mice.
description
1991 nî lūn-bûn
@nan
1991年の論文
@ja
1991年学术文章
@wuu
1991年学术文章
@zh
1991年学术文章
@zh-cn
1991年学术文章
@zh-hans
1991年学术文章
@zh-my
1991年学术文章
@zh-sg
1991年學術文章
@yue
1991年學術文章
@zh-hant
name
A gene(s) within the H-2D regi ...... oplasmic encephalitis in mice.
@en
type
label
A gene(s) within the H-2D regi ...... oplasmic encephalitis in mice.
@en
prefLabel
A gene(s) within the H-2D regi ...... oplasmic encephalitis in mice.
@en
P2093
P1433
P1476
A gene(s) within the H-2D regi ...... oplasmic encephalitis in mice.
@en
P2093
P304
P577
1991-12-01T00:00:00Z