Anomalous levels of Cl- transporters cause a decrease of GABAergic inhibition in human peritumoral epileptic cortex.
about
Long-range and short-range tumor-stroma networks synergistically contribute to tumor-associated epilepsy.Expanding Spectrum of Sodium Potassium Chloride Co-transporters in the Pathophysiology of DiseasesEpilepsy related to developmental tumors and malformations of cortical developmentCurrent view on the functional regulation of the neuronal K(+)-Cl(-) cotransporter KCC2GABAergic disinhibition and impaired KCC2 cotransporter activity underlie tumor-associated epilepsy.Novel treatment targets for cerebral edemaAstaxanthin protects astrocytes against trauma-induced apoptosis through inhibition of NKCC1 expression via the NF-κB signaling pathwayAnomalous expression of chloride transporters in the sclerosed hippocampus of mesial temporal lobe epilepsy patientsA role for intracellular zinc in glioma alteration of neuronal chloride equilibrium.Cortical GABAergic excitation contributes to epileptic activities around human glioma.WONOEP appraisal: Development of epilepsy biomarkers-What we can learn from our patients?Changes in expression of the chloride homeostasis-regulating genes, KCC2 and NKCC1, in the blood of cirrhotic patients with hepatic encephalopathy.Epilepsy in glioma patients: mechanisms, management, and impact of anticonvulsant therapyLong-term epilepsy-associated tumors.Epilepsy in glioblastoma patients: basic mechanisms and current problems in treatment.Tumoral epileptogenicity: how does it happen?Optimizing antiepileptic drug treatment in tumoral epilepsy.Peritumoral epilepsy: relating form and function for surgical success.GABAergic signalling in the immune system.Exploring novel AEDs from drugs used for treatment of non-epileptic disorders.Epileptic seizures in anaplastic gangliogliomas.Compromised GABAergic inhibition contributes to tumor-associated epilepsy.Transient increase in neuronal chloride concentration by neuroactive aminoacids released from glioma cells.Enhanced expression of potassium-chloride cotransporter KCC2 in human temporal lobe epilepsy.Pumilio2-deficient mice show a predisposition for epilepsy.Pro-Brain-Derived Neurotrophic Factor (proBDNF)-Mediated p75NTR Activation Promotes Depolarizing Actions of GABA and Increases Susceptibility to Epileptic Seizures.Fractalkine/CX3CL1 modulates GABAA currents in human temporal lobe epilepsy.The KCC2 Cotransporter and Human Epilepsy: Getting Excited About Inhibition.
P2860
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P2860
Anomalous levels of Cl- transporters cause a decrease of GABAergic inhibition in human peritumoral epileptic cortex.
description
2011 nî lūn-bûn
@nan
2011年の論文
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2011年学术文章
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2011年学术文章
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2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
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2011年學術文章
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2011年學術文章
@zh-hant
name
Anomalous levels of Cl- transp ...... peritumoral epileptic cortex.
@en
Anomalous levels of Cl- transp ...... peritumoral epileptic cortex.
@nl
type
label
Anomalous levels of Cl- transp ...... peritumoral epileptic cortex.
@en
Anomalous levels of Cl- transp ...... peritumoral epileptic cortex.
@nl
prefLabel
Anomalous levels of Cl- transp ...... peritumoral epileptic cortex.
@en
Anomalous levels of Cl- transp ...... peritumoral epileptic cortex.
@nl
P2093
P2860
P1433
P1476
Anomalous levels of Cl- transp ...... peritumoral epileptic cortex.
@en
P2093
Clotilde Lauro
Cristina Limatola
Cristina Roseti
Luca Conti
Marjolein de Groot
Raffaela Cipriani
P2860
P304
P356
10.1111/J.1528-1167.2011.03111.X
P577
2011-06-02T00:00:00Z