Acetazolamide and midazolam act synergistically to inhibit neuropathic pain.
about
Non-Classical Inhibition of Carbonic AnhydraseInhibitory synaptic plasticity: spike timing-dependence and putative network function.Probing the surface of human carbonic anhydrase for clues towards the design of isoform specific inhibitorsPotential of GABA-ergic cell therapy for schizophrenia, neuropathic pain, and Alzheimer's and Parkinson's diseases.Acetazolamide attenuates chemical-stimulated but not thermal-stimulated acute pain in mice.Cation-chloride cotransporters in neuronal development, plasticity and diseaseEfficacy of synaptic inhibition depends on multiple, dynamically interacting mechanisms implicated in chloride homeostasis.Modulation of spinal GABAergic analgesia by inhibition of chloride extrusion capacity in mice.The AMPK Activator A769662 Blocks Voltage-Gated Sodium Channels: Discovery of a Novel Pharmacophore with Potential Utility for Analgesic Development.Morphine hyperalgesia gated through microglia-mediated disruption of neuronal Cl⁻ homeostasis.P2X4R+ microglia drive neuropathic pain."Bedside-to-Bench" Behavioral Outcomes in Animal Models of Pain: Beyond the Evaluation of Reflexes.Inhibition of carbonic anhydrase augments GABAA receptor-mediated analgesia via a spinal mechanism of action.Molecules in pain and sex: a developing story.Carbonic anhydrase inhibitors and activators for novel therapeutic applications.The neurobiology of opiate motivation.Inhibitory regulation of the pain gate and how its failure causes pathological pain.Acetazolamide reduces postoperative pain following laparoscopic inguinal herniorrhaphy.Glycine receptors and glycine transporters: targets for novel analgesics?Drug interaction considerations in the therapeutic use of carbonic anhydrase inhibitors.Synaptic conversion of chloride-dependent synapses in spinal nociceptive circuits: roles in neuropathic pain.Carbonic anhydrase inhibition and the management of neuropathic pain.Mild KCC2 Hypofunction Causes Inconspicuous Chloride Dysregulation that Degrades Neural Coding.In vivo and in vitro protective effects of omeprazole against neuropathic pain.Forebrain GABAergic neuron precursors integrate into adult spinal cord and reduce injury-induced neuropathic pain.Neuroligin 2 regulates spinal GABAergic plasticity in hyperalgesic priming, a model of the transition from acute to chronic painIntrinsic thermodynamics of trifluoromethanesulfonamide and ethoxzolamide binding to human carbonic anhydrase VII.Anomalous levels of Cl- transporters cause a decrease of GABAergic inhibition in human peritumoral epileptic cortex.Identification of potent and selective human carbonic anhydrase VII (hCA VII) inhibitors.The Crystal Structure of a hCA VII Variant Provides Insights into the Molecular Determinants Responsible for Its Catalytic BehaviorUltra-diluted Toxicodendron pubescens attenuates pro-inflammatory cytokines and ROS- mediated neuropathic pain in ratsPyridinium derivatives of histamine are potent activators of cytosolic carbonic anhydrase isoforms I, II and VII
P2860
Q26738744-64689988-B1B3-4D9D-A3F3-DF5F7A3FA793Q26865106-48C2C598-FA46-4426-8471-913C7573EBA2Q28085120-694C102A-85C1-486E-9C4F-591DD0B1B544Q30357529-EE756C21-BC48-42CE-BED9-4BE6297A53D7Q33823701-0413433F-C593-471A-8CBD-6789BB805366Q34959293-CB93267D-1B1A-4846-87A0-42EBF1E13C38Q35203892-1B2F44EF-1C10-43C9-B619-EB9C4F34B994Q36007778-C266F704-FD08-499B-9CEF-C5CFDBE5235CQ36258703-FFA0D0E9-2E32-45FA-818C-082C6B67DC0CQ37152999-3394FED0-FD11-482D-86CA-42361AF71BC3Q37256253-58617035-8759-4F2C-9E72-C2D060182511Q37362035-9CDA502D-44E7-46E1-816F-E222D92B58B7Q37678668-8946804D-78D7-43FB-8D95-011DAA5115A3Q37686434-40ED3C43-3127-43CF-8BB6-7E378B71BA86Q37909406-9DDA6D02-3A66-4E24-8BE2-08416F9FF9BAQ38048764-3F2DC474-3F4A-4230-AD04-37B7F66CC1B4Q38365447-5875F996-6CB5-41AC-88D8-69CBA8240DEBQ38612744-E360E500-2AB4-4319-BAA6-18D1A949E15AQ38632864-1453938B-4A78-441F-929C-10A5F7CC5E0EQ38733892-C6EE7A34-239C-4BB1-AA5B-7F2F71FDD8D3Q38737690-4AF88F67-C43C-43B1-9F9A-51D291060130Q38841249-2F3698C3-B91C-41FB-B01E-CAD5CE500A32Q41826853-718BF21F-656B-437D-B4F2-312444E8009AQ41830886-35B0F419-5378-4D19-81EC-0F646DBBF251Q42178613-CBD62FB3-C4ED-42E8-9FB9-8D2213D73302Q42541482-2C3ED690-0AC7-4513-B1EF-FD0E50B1DEDFQ48343823-F32C5F0D-053B-49B9-A39F-7F6BA8655191Q48671988-8056C96A-ACAB-4FB3-BFA1-3BCFAD03FC5BQ50336076-D0515D9F-AECB-4EC8-90BC-C4AEFD1795B8Q57600742-36F6C96F-FC16-46AA-85E0-6944E1149583Q58756363-AAD15487-411C-42D2-B25E-40086791EB07Q58863220-BE2A0D57-288D-45A4-BEDE-BE85CCD143C3
P2860
Acetazolamide and midazolam act synergistically to inhibit neuropathic pain.
description
2009 nî lūn-bûn
@nan
2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Acetazolamide and midazolam act synergistically to inhibit neuropathic pain.
@ast
Acetazolamide and midazolam act synergistically to inhibit neuropathic pain.
@en
Acetazolamide and midazolam act synergistically to inhibit neuropathic pain.
@nl
type
label
Acetazolamide and midazolam act synergistically to inhibit neuropathic pain.
@ast
Acetazolamide and midazolam act synergistically to inhibit neuropathic pain.
@en
Acetazolamide and midazolam act synergistically to inhibit neuropathic pain.
@nl
prefLabel
Acetazolamide and midazolam act synergistically to inhibit neuropathic pain.
@ast
Acetazolamide and midazolam act synergistically to inhibit neuropathic pain.
@en
Acetazolamide and midazolam act synergistically to inhibit neuropathic pain.
@nl
P2860
P1433
P1476
Acetazolamide and midazolam act synergistically to inhibit neuropathic pain.
@en
P2093
Marina Asiedu
Michael H Ossipov
P2860
P304
P356
10.1016/J.PAIN.2009.11.015
P407
P577
2009-12-14T00:00:00Z