IL-4 is protective against development of toxoplasmic encephalitis.
about
Recombinant Toxoplasma gondii phosphoglycerate mutase 2 confers protective immunity against toxoplasmosis in BALB/c miceInterferon-gamma- and perforin-mediated immune responses for resistance against Toxoplasma gondii in the brainToxoplasma gondii soluble tachyzoite antigen triggers protective mechanisms against fatal intestinal pathology in oral infection of C57BL/6 miceMAP kinase phosphatase-2 plays a key role in the control of infection with Toxoplasma gondii by modulating iNOS and arginase-1 activities in miceMast cells modulate acute toxoplasmosis in murine modelsNeurological and behavioral abnormalities, ventricular dilatation, altered cellular functions, inflammation, and neuronal injury in brains of mice due to common, persistent, parasitic infection.Induction of interleukin-4 (IL-4) by legionella pneumophila infection in BALB/c mice and regulation of tumor necrosis factor alpha, IL-6, and IL-1beta.Removal of Toxoplasma gondii cysts from the brain by perforin-mediated activity of CD8+ T cells.Acute and chronic phases of Toxoplasma gondii infection in mice modulate the host immune responses.Immunization of BALB/c mice with killed Neospora caninum tachyzoite antigen induces a type 2 immune response and exacerbates encephalitis and neurological diseaseIn the absence of endogenous gamma interferon, mice acutely infected with Neospora caninum succumb to a lethal immune response characterized by inactivation of peritoneal macrophages.Protective role for interleukin-5 during chronic Toxoplasma gondii infection.Counter-protective role for interleukin-5 during acute Toxoplasma gondii infectionRequirement of non-T cells that produce gamma interferon for prevention of reactivation of Toxoplasma gondii infection in the brainBoth Th1 and Th2 cytokines affect the ability of monoclonal antibodies to protect mice against Cryptococcus neoformans.Protective Toxoplasma gondii-specific T-cell responses require T-cell-specific expression of protein kinase C-theta.Central role for interleukin-4 in regulating nitric oxide-mediated inhibition of T-cell proliferation and gamma interferon production in schistosomiasisProtective immunity induced by peptides of AMA1, RON2 and RON4 containing T-and B-cell epitopes via an intranasal route against toxoplasmosis in mice.Different strains of Toxoplasma gondii induce different cytokine responses in CBA/Ca miceMechanisms of innate resistance to Toxoplasma gondii infection.Impaired resistance to the development of toxoplasmic encephalitis in interleukin-6-deficient mice.Gamma interferon induces Fas-dependent apoptosis of Peyer's patch T cells in mice following peroral infection with Toxoplasma gondii.Inducible nitric oxide is essential for host control of persistent but not acute infection with the intracellular pathogen Toxoplasma gondiiEndogenous interleukin 4 is required for development of protective CD4+ T helper type 1 cell responses to Candida albicans.Gamma interferon production, but not perforin-mediated cytolytic activity, of T cells is required for prevention of toxoplasmic encephalitis in BALB/c mice genetically resistant to the disease.IL-4 receptor-alpha-dependent control of Cryptococcus neoformans in the early phase of pulmonary infection.Immune responses against protozoan parasites: a focus on the emerging role of Nod-like receptors.STAT6 signalling is important in CD8 T-cell activation and defence against Toxoplasma gondii infection in the brain.
P2860
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P2860
IL-4 is protective against development of toxoplasmic encephalitis.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年学术文章
@wuu
1996年学术文章
@zh
1996年学术文章
@zh-cn
1996年学术文章
@zh-hans
1996年学术文章
@zh-my
1996年学术文章
@zh-sg
1996年學術文章
@yue
1996年學術文章
@zh-hant
name
IL-4 is protective against development of toxoplasmic encephalitis.
@en
IL-4 is protective against development of toxoplasmic encephalitis.
@nl
type
label
IL-4 is protective against development of toxoplasmic encephalitis.
@en
IL-4 is protective against development of toxoplasmic encephalitis.
@nl
prefLabel
IL-4 is protective against development of toxoplasmic encephalitis.
@en
IL-4 is protective against development of toxoplasmic encephalitis.
@nl
P2093
P1476
IL-4 is protective against development of toxoplasmic encephalitis
@en
P2093
Liesenfeld O
Remington JS
P304
P407
P577
1996-09-01T00:00:00Z