Transgenic mice overexpressing glutathione peroxidase are resistant to myocardial ischemia reperfusion injury.
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The nature of antioxidant defense mechanisms: a lesson from transgenic studiesSelenium and its supplementation in cardiovascular disease--what do we know?Paradoxical Roles of Antioxidant Enzymes: Basic Mechanisms and Health ImplicationsHypoxia and acidosis activate cardiac myocyte death through the Bcl-2 family protein BNIP3.Impaired angiogenesis in glutathione peroxidase-1-deficient mice is associated with endothelial progenitor cell dysfunctionCardioprotective mechanism of diazoxide involves the inhibition of succinate dehydrogenaseTransgenic A1 adenosine receptor overexpression increases myocardial resistance to ischemiaEmerging role for antioxidant therapy in protection against diabetic cardiac complications: experimental and clinical evidence for utilization of classic and new antioxidantsSulfaphenazole protects heart against ischemia-reperfusion injury and cardiac dysfunction by overexpression of iNOS, leading to enhancement of nitric oxide bioavailability and tissue oxygenation.Importance of the bioenergetic reserve capacity in response to cardiomyocyte stress induced by 4-hydroxynonenal.The role of Nrf2-mediated pathway in cardiac remodeling and heart failure.Antioxidant network expression abrogates oxidative posttranslational modifications in mice.Transgenic and knockout models for studying the role of lung antioxidant enzymes in defense against hyperoxia.Impairment of synaptic transmission by transient hypoxia in hippocampal slices: improved recovery in glutathione peroxidase transgenic mice.Myocardial ischaemia inhibits mitochondrial metabolism of 4-hydroxy-trans-2-nonenal.Models of reactive oxygen species in cancer.Genetic Deficiency of Glutathione S-Transferase P Increases Myocardial Sensitivity to Ischemia-Reperfusion Injury.CUEDC2 modulates cardiomyocyte oxidative capacity by regulating GPX1 stability.Prevention of ischemia/reperfusion-induced cardiac apoptosis and injury by melatonin is independent of glutathione peroxdiase 1.Effects of downregulation of microRNA-181a on H2O2-induced H9c2 cell apoptosis via the mitochondrial apoptotic pathway.Tetracyclines: a pleitropic family of compounds with promising therapeutic properties. Review of the literature.The role of succinate and ROS in reperfusion injury - A critical appraisal.Targeted disruption of the glutaredoxin 1 gene does not sensitize adult mice to tissue injury induced by ischemia/reperfusion and hyperoxia.Alginate oligosaccharide alleviates myocardial reperfusion injury by inhibiting nitrative and oxidative stress and endoplasmic reticulum stress-mediated apoptosis.Ischemic-reperfused isolated working mouse hearts: membrane damage and type IIA phospholipase A2.Activity staining of glutathione peroxidase after electrophoresis on native and sodium dodecyl sulfate polyacrylamide gels.H(2)O(2) opens mitochondrial K(ATP) channels and inhibits GABA receptors via protein kinase C-epsilon in cardiomyocytes.Role of intracellular antioxidant enzymes after in vivo myocardial ischemia and reperfusion.Protective effect of melatonin on myocardial infarction.Depletion of antioxidants is associated with no-reflow phenomenon in acute myocardial infarction.Redox status of patients before cardiac surgery.Glutathione peroxidases as oncotargets.Lung Ischaemia-Reperfusion Injury: The Role of Reactive Oxygen Species.Metabolic syndrome and selenium during gestation and lactation.C-phycocyanin protects against ischemia-reperfusion injury of heart through involvement of p38 MAPK and ERK signaling.Selenoproteins and cardiovascular stress.Preoperative fasting protects mice against hepatic ischemia/reperfusion injury: mechanisms and effects on liver regeneration.Targeted disruption of peroxiredoxin 6 gene renders the heart vulnerable to ischemia-reperfusion injury.Cardioprotection by K(ATP) channels in wild-type hearts and hearts overexpressing A(1)-adenosine receptors.Dunye Guanxinning Improves Acute Myocardial Ischemia-Reperfusion Injury by Inhibiting Neutrophil Infiltration and Caspase-1 Activity.
P2860
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P2860
Transgenic mice overexpressing glutathione peroxidase are resistant to myocardial ischemia reperfusion injury.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年学术文章
@wuu
1996年学术文章
@zh
1996年学术文章
@zh-cn
1996年学术文章
@zh-hans
1996年学术文章
@zh-my
1996年学术文章
@zh-sg
1996年學術文章
@yue
1996年學術文章
@zh-hant
name
Transgenic mice overexpressing ...... l ischemia reperfusion injury.
@en
Transgenic mice overexpressing ...... l ischemia reperfusion injury.
@nl
type
label
Transgenic mice overexpressing ...... l ischemia reperfusion injury.
@en
Transgenic mice overexpressing ...... l ischemia reperfusion injury.
@nl
prefLabel
Transgenic mice overexpressing ...... l ischemia reperfusion injury.
@en
Transgenic mice overexpressing ...... l ischemia reperfusion injury.
@nl
P2093
P356
P1476
Transgenic mice overexpressing ...... l ischemia reperfusion injury.
@en
P2093
Engelman DT
Engelman RM
Oberley TD
Watanabe M
P304
P356
10.1006/JMCC.1996.0165
P577
1996-08-01T00:00:00Z