Sulfaphenazole protects heart against ischemia-reperfusion injury and cardiac dysfunction by overexpression of iNOS, leading to enhancement of nitric oxide bioavailability and tissue oxygenation.
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Clinical EPR: unique opportunities and some challengesS-nitrosoglutathione accelerates recovery from 5-fluorouracil-induced oral mucositisMolecular packing and magnetic properties of lithium naphthalocyanine crystals: hollow channels enabling permeability and paramagnetic sensitivity to molecular oxygen.Autophagy and protein kinase C are required for cardioprotection by sulfaphenazole.Endothelial NOS activity and myocardial oxygen metabolism define the salvageable ischemic time window for ischemic postconditioning.Theory, instrumentation, and applications of electron paramagnetic resonance oximetry.Trimetazidine, administered at the onset of reperfusion, ameliorates myocardial dysfunction and injury by activation of p38 mitogen-activated protein kinase and Akt signaling.Opening of the mitoKATP channel and decoupling of mitochondrial complex II and III contribute to the suppression of myocardial reperfusion hyperoxygenation.Cardioprotection by HO-4038, a novel verapamil derivative, targeted against ischemia and reperfusion-mediated acute myocardial infarctionIntermittent hypoxia exacerbates pancreatic β-cell dysfunction in a mouse model of diabetes mellitusOxygen and oxygenation in stem-cell therapy for myocardial infarctionFunctional electron paramagnetic resonance imaging of ischemic rat heart: Monitoring of tissue oxygenation and pH.Selective inhibition of hypoxia-inducible factor 1α ameliorates adipose tissue dysfunction.Oxygen cycling in conjunction with stem cell transplantation induces NOS3 expression leading to attenuation of fibrosis and improved cardiac functionCarbon monoxide increases inducible NOS expression that mediates CO-induced myocardial damage during ischemia-reperfusion.Electron paramagnetic resonance monitoring of ischemia-induced myocardial oxygen depletion and acidosis in isolated rat hearts using soluble paramagnetic probes.Loss of metallothionein predisposes mice to diethylnitrosamine-induced hepatocarcinogenesis by activating NF-kappaB target genes.Pharmacological preconditioning of mesenchymal stem cells with trimetazidine (1-[2,3,4-trimethoxybenzyl]piperazine) protects hypoxic cells against oxidative stress and enhances recovery of myocardial function in infarcted heart through Bcl-2 expressThe preconditioning pulmonary protective effect of volatile isoflurane in acute lung injury is mediated by activation of endogenous iNOS.Supplemental Oxygen Protects Heart Against Acute Myocardial Infarction
P2860
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P2860
Sulfaphenazole protects heart against ischemia-reperfusion injury and cardiac dysfunction by overexpression of iNOS, leading to enhancement of nitric oxide bioavailability and tissue oxygenation.
description
2009 nî lūn-bûn
@nan
2009 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Sulfaphenazole protects heart ...... bility and tissue oxygenation.
@ast
Sulfaphenazole protects heart ...... bility and tissue oxygenation.
@en
type
label
Sulfaphenazole protects heart ...... bility and tissue oxygenation.
@ast
Sulfaphenazole protects heart ...... bility and tissue oxygenation.
@en
prefLabel
Sulfaphenazole protects heart ...... bility and tissue oxygenation.
@ast
Sulfaphenazole protects heart ...... bility and tissue oxygenation.
@en
P2093
P2860
P356
P1476
Sulfaphenazole protects heart ...... bility and tissue oxygenation.
@en
P2093
Iyyapu K Mohan
Mahmood Khan
Narasimham L Parinandi
Periannan Kuppusamy
Robert L Hamlin
Sainath R Kotha
Vijay K Kutala
P2860
P304
P356
10.1089/ARS.2008.2155
P577
2009-04-01T00:00:00Z