Prolonged inhaled allergen exposure can induce persistent tolerance. - Wikidata - awgldk - TriplyDB

Prolonged inhaled allergen exposure can induce persistent tolerance.

Prolonged inhaled allergen exposure can induce persistent tolerance.

http://www.wikidata.org/entity/Q49064763

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Compartmental and temporal dynamics of chronic inflammation and airway remodelling in a chronic asthma mouse model Barriers to inhaled gene therapy of obstructive lung diseases: A review Developments in the field of allergy mechanisms in 2015 through the eyes of Clinical & Experimental Allergy.Regulation of the T helper cell type 2 (Th2)/T regulatory cell (Treg) balance by IL-4 and STAT6 Alveolar macrophages play a key role in cockroach-induced allergic inflammation via TNF-α pathway.β5i subunit deficiency of the immunoproteasome leads to reduced Th2 response in OVA induced acute asthma.Re-challenge with ovalbumin failed to induce bronchial asthma in mice with eosinophilic bronchitis.Mucosal exposure to cockroach extract induces allergic sensitization and allergic airway inflammation.Long-Term Exposure to House Dust Mite Leads to the Suppression of Allergic Airway Disease Despite Persistent Lung Inflammation Early-life co-administration of cockroach allergen and endotoxin augments pulmonary and systemic responses.Monitoring inflammation and airway remodeling by fluorescence molecular tomography in a chronic asthma model.Regulatory B cells from hilar lymph nodes of tolerant mice in a murine model of allergic airway disease are CD5+, express TGF-β, and co-localize with CD4+Foxp3+ T cells.Different effects of deep inspirations on central and peripheral airways in healthy and allergen-challenged mice.Tissue-resident dendritic cells and diseases involving dendritic cell malfunction CD4⁺ and CD8⁺ T cells play a central role in a HDM driven model of allergic asthma Subcutaneous late phase responses are augmented during local inhalational tolerance in a murine asthma model Regulatory role of B cells in a murine model of allergic airway disease.Acute exercise decreases airway inflammation, but not responsiveness, in an allergic asthma model.Combined sensitization of mice to extracts of dust mite, ragweed, and Aspergillus species breaks through tolerance and establishes chronic features of asthma.Comparison of adjuvant and adjuvant-free murine experimental asthma models Where asthma and hypersensitivity pneumonitis meet and differ: noneosinophilic severe asthma.Innate immunity in allergic disease.Exposure of immunologically naive laboratory rodents to antigen via the airways. Where does tolerance stop and sensitization begin?Key regulators of sensitization and tolerance: GM-CSF, IL-10, TGF-β and the Notch signaling pathway in adjuvant-free experimental models of respiratory allergy.Regulatory tone and mucosal immunity in asthma Cigarette smoke enhances Th-2 driven airway inflammation and delays inhalational tolerance.Priming with high and low respiratory allergen dose induces differential CD4+ T helper type 2 cells and IgE/IgG1 antibody responses in mice.Airway epithelial NF-κB activation promotes the ability to overcome inhalational antigen tolerance Amelioration of ovalbumin-induced allergic airway disease following Der p 1 peptide immunotherapy is not associated with induction of IL-35.Tumor necrosis factor-related apoptosis-inducing ligand mediates the resolution of allergic airway inflammation induced by chronic allergen inhalation.Antigen-induced airway hyperresponsiveness in absence of broncho-obstruction in sensitized guinea pigs.Therapeutic vaccines for allergic disease.Proposed short-term model of acute allergic response, without adjuvant use, in the lungs of mice.Prenatal allergen exposures prevent allergen-induced sensitization and airway inflammation in young mice.A novel prostacyclin agonist protects against airway hyperresponsiveness and remodeling in mice.

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P2860

Prolonged inhaled allergen exposure can induce persistent tolerance.

http://www.wikidata.org/entity/Q49064763

description

2007 nî lūn-bûn

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2007年の論文

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年學術文章

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2007年學術文章

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Prolonged inhaled allergen exposure can induce persistent tolerance.

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Prolonged inhaled allergen exposure can induce persistent tolerance.

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Prolonged inhaled allergen exposure can induce persistent tolerance.

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Prolonged inhaled allergen exposure can induce persistent tolerance.

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Prolonged inhaled allergen exposure can induce persistent tolerance.

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Prolonged inhaled allergen exposure can induce persistent tolerance.

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American Journal of Respiratory Cell and Molecular Biology

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Prolonged inhaled allergen exposure can induce persistent tolerance.

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Chris L Van Hove

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Guy F Joos

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Kurt G Tournoy

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P2860

Antigen-specific regulatory T cells develop via the ICOS-ICOS-ligand pathway and inhibit allergen-induced airway hyperreactivity

Engagement of the PD-1 immunoinhibitory receptor by a novel B7 family member leads to negative regulation of lymphocyte activation

Environmental exposure to endotoxin and its relation to asthma in school-age children

Requirement of CD4-positive T cells for cellular recruitment to the lungs of mice in response to a particulate intratracheal antigen

Modeling allergic asthma in mice: pitfalls and opportunities.

Chronic inhaled ovalbumin exposure induces antigen-dependent but not antigen-specific inhalational tolerance in a murine model of allergic airway disease.

Murine models of asthma.

Chronic airway hyperreactivity, goblet cell hyperplasia, and peribronchial fibrosis during allergic airway disease induced by Aspergillus fumigatus.

Tissue expression of PD-L1 mediates peripheral T cell tolerance.

Regulatory T cells and IL-10 in allergic inflammation.

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