Endogenously produced substance P contributes to lymphocyte proliferation induced by dendritic cells and direct TCR ligation.
about
Neuroimmunomodulation in the Gut: Focus on Inflammatory Bowel DiseaseCentral mediators involved in the febrile response: effects of antipyretic drugsImmunologists getting nervous: neuropeptides, dendritic cells and T cell activationInduction of tachykinin production in airway epithelia in response to viral infectionImmunological role of neuronal receptor vanilloid receptor 1 expressed on dendritic cells.Neural mechanisms in asthma.Implications of non-canonical G-protein signaling for the immune systemThe dendritic cell in allergic airway diseases: a new player to the game.Neutralizing anti-F glycoprotein and anti-substance P antibody treatment effectively reduces infection and inflammation associated with respiratory syncytial virus infection.Involvement of substance P and the NK-1 receptor in human pathology.Loss of innervation and axon plasticity accompanies impaired diabetic wound healing.Cross-talk between human mast cells and bronchial epithelial cells in plasminogen activator inhibitor-1 production via transforming growth factor-β1.Substance P in the corneal stroma regulates the severity of herpetic stromal keratitis lesions.Mechanisms of selection mediated by interleukin-7, the preBCR, and hemokinin-1 during B-cell development.Substance P is responsible for physiological alterations such as increased chloride ion secretion and glucose malabsorption in cryptosporidiosis.NEUROPEPTIDES AND NEUROGENIC MECHANISMS IN ORAL AND PERIODONTAL INFLAMMATION.Oral inflammatory diseases and systemic inflammation: role of the macrophage.Pulmonary dendritic cells.An involvement of neurokinin-1 receptor in FcεRΙ-mediated RBL-2H3 mast cell activation.Immunomodulatory properties of substance P: the gastrointestinal system as a model.Interleukin-12 (IL-12) and IL-23 induction of substance p synthesis in murine T cells and macrophages is subject to IL-10 and transforming growth factor beta regulation.Inflammation and neuropeptides: the connection in diabetic wound healing.Substance P downregulates expression of the high affinity IgE receptor (FcepsilonRI) by human mast cellsProinflammatory tachykinins that signal through the neurokinin 1 receptor promote survival of dendritic cells and potent cellular immunity.Activation of neurokinin-1 receptors up-regulates substance P and neurokinin-1 receptor expression in murine pancreatic acinar cells.Effect of Corneal Nerve Ablation on Immune Tolerance Induced by Corneal Allografts, Oral Immunization, or Anterior Chamber Injection of Antigens.Tachykinins and their receptors: contributions to physiological control and the mechanisms of disease.Significance of Conversation between Mast Cells and Nerves.Dendritic cells and airway epithelial cells at the interface between innate and adaptive immune responses.Direct and indirect antimicrobial activities of neuropeptides and their therapeutic potential.Inflammatory and immune pathways in the pathogenesis of periodontal disease.Relationship between levels of neuropeptide Substance P in periodontal disease and chronic pain: a literature review.Substance P and the regulation of inflammation in infections and inflammatory bowel disease.Identification and putative roles of distinct subtypes of intestinal dendritic cells in neuroimmune communication: what can be learned from other organ systems?Gastrointestinal neuroendocrine peptides/amines in inflammatory bowel disease.Neuropeptide substance P and the immune response.Role of tachykinins in the host response to murine gammaherpesvirus infection.Substance P primes lipoteichoic acid- and Pam3CysSerLys4-mediated activation of human mast cells by up-regulating Toll-like receptor 2.Role of the tachykinin NK1 receptor in a murine model of cigarette smoke-induced pulmonary inflammation.Role of Substance P Neuropeptide in Inflammation, Wound Healing, and Tissue Homeostasis.
P2860
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P2860
Endogenously produced substance P contributes to lymphocyte proliferation induced by dendritic cells and direct TCR ligation.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
@yue
1999年學術文章
@zh-hant
name
Endogenously produced substanc ...... cells and direct TCR ligation.
@en
Endogenously produced substanc ...... cells and direct TCR ligation.
@nl
type
label
Endogenously produced substanc ...... cells and direct TCR ligation.
@en
Endogenously produced substanc ...... cells and direct TCR ligation.
@nl
prefLabel
Endogenously produced substanc ...... cells and direct TCR ligation.
@en
Endogenously produced substanc ...... cells and direct TCR ligation.
@nl
P2093
P2860
P1476
Endogenously produced substanc ...... cells and direct TCR ligation.
@en
P2093
Carro-Muino I
De Veerman M
Everaert EG
Germonpré PR
Lambrecht BN
Pauwels RA
Thielemans K
de Felipe C
P2860
P304
P356
10.1002/(SICI)1521-4141(199912)29:12<3815::AID-IMMU3815>3.0.CO;2-#
P407
P577
1999-12-01T00:00:00Z