Glutamate-induced and NMDA receptor-mediated neurodegeneration entails P2Y1 receptor activation.

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Glutamate-induced and NMDA receptor-mediated neurodegeneration entails P2Y1 receptor activation.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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Glutamate-induced and NMDA rec ...... ails P2Y1 receptor activation.

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Glutamate-induced and NMDA rec ...... ails P2Y1 receptor activation.

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Glutamate-induced and NMDA rec ...... ails P2Y1 receptor activation.

@en

Glutamate-induced and NMDA rec ...... ails P2Y1 receptor activation.

@nl

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Glutamate-induced and NMDA rec ...... ails P2Y1 receptor activation.

@en

Glutamate-induced and NMDA rec ...... ails P2Y1 receptor activation.

@nl

P1476

Glutamate-induced and NMDA rec ...... tails P2Y1 receptor activation

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Ben A Bahr

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Carla G Silva

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Daniela Pochmann

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Juan Lerma

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Lisiane O Porciúncula

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Rui O Beleza

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P2860

The pathogenic activation of calpain: a marker and mediator of cellular toxicity and disease states

Chronic Glutamate Toxicity in Neurodegenerative Diseases-What is the Evidence?

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Dual presynaptic control by ATP of glutamate release via facilitatory P2X1, P2X2/3, and P2X3 and inhibitory P2Y1, P2Y2, and/or P2Y4 receptors in the rat hippocampus

Extrasynaptic NMDARs oppose synaptic NMDARs by triggering CREB shut-off and cell death pathways

Inhibition of N-type voltage-activated calcium channels in rat dorsal root ganglion neurons by P2Y receptors is a possible mechanism of ADP-induced analgesia.

Calpain product of WT-CRMP2 reduces the amount of surface NR2B NMDA receptor subunit.

Excitatory amino acids as a final common pathway for neurologic disorders.

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scholarly article

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10.1038/S41419-018-0351-1

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2018-02-20T00:00:00Z

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29463792

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5833818

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