Substance P and bradykinin stimulate plasma extravasation in the mouse gastrointestinal tract and pancreas.
about
The role of Transient Receptor Potential Vanilloid 1 (TRPV1) channels in pancreatitisA role for transient receptor potential vanilloid 4 in tonicity-induced neurogenic inflammation.Substance P mediates inflammatory oedema in acute pancreatitis via activation of the neurokinin-1 receptor in rats and miceActions of bradykinin on electrical and synaptic behavior of neurones in the myenteric plexus of guinea-pig small intestineLeukotriene B4 mediates inflammation via TRPV1 in duct obstruction-induced pancreatitis in rats.Neutral endopeptidase (EC 3.4.24.11) terminates colitis by degrading substance PEndothelin-converting enzyme 1 degrades neuropeptides in endosomes to control receptor recycling.Energetic etiologies of acute pancreatitis: A report of five cases.Role of neurogenic inflammation in pancreatitis and pancreatic pain.The RNA-binding protein quaking maintains endothelial barrier function and affects VE-cadherin and β-catenin protein expression.Pro-inflammatory effects of hydrogen sulphide on substance P in caerulein-induced acute pancreatitis.Tachykinins and their receptors: contributions to physiological control and the mechanisms of disease.Substance P-stimulated interleukin-8 expression in human colonic epithelial cells involves Rho family small GTPases.Loss of a Candidate Biliary Atresia Susceptibility Gene, add3a, Causes Biliary Developmental Defects in Zebrafish.Neurogenic plasma leakage in mouse airways.Neutral endopeptidase (EC 3.4.24.11) downregulates the onset of intestinal inflammation in the nematode infected mouse.A pH-sensitive, neurogenic pathway mediates disease severity in a model of post-ERCP pancreatitis.Deletion of neutral endopeptidase exacerbates intestinal inflammation induced by Clostridium difficile toxin A.Capsaicin vanilloid receptor-1 mediates substance P release in experimental pancreatitis.Primary sensory neurons: a common final pathway for inflammation in experimental pancreatitis in rats.Preprotachykinin-A gene deletion protects mice against acute pancreatitis and associated lung injury.Attenuation of acute experimental colitis by preventing NPY Y1 receptor signaling.NK-1 receptor desensitization and neutral endopeptidase terminate SP-induced pancreatic plasma extravasation.
P2860
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P2860
Substance P and bradykinin stimulate plasma extravasation in the mouse gastrointestinal tract and pancreas.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年学术文章
@wuu
1997年学术文章
@zh
1997年学术文章
@zh-cn
1997年学术文章
@zh-hans
1997年学术文章
@zh-my
1997年学术文章
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1997年學術文章
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1997年學術文章
@zh-hant
name
Substance P and bradykinin sti ...... intestinal tract and pancreas.
@en
Substance P and bradykinin sti ...... intestinal tract and pancreas.
@nl
type
label
Substance P and bradykinin sti ...... intestinal tract and pancreas.
@en
Substance P and bradykinin sti ...... intestinal tract and pancreas.
@nl
prefLabel
Substance P and bradykinin sti ...... intestinal tract and pancreas.
@en
Substance P and bradykinin sti ...... intestinal tract and pancreas.
@nl
P2093
P1476
Substance P and bradykinin sti ...... intestinal tract and pancreas.
@en
P2093
C Emanueli
K Kirkwood
P Geppetti
P304
P356
10.1152/AJPGI.1997.272.4.G785
P407
P433
P577
1997-04-01T00:00:00Z