Neutral endopeptidase (EC 3.4.24.11) terminates colitis by degrading substance P
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Endothelin-converting enzyme-1 regulates endosomal sorting of calcitonin receptor-like receptor and beta-arrestinsThe Neuromodulation of the Intestinal Immune System and Its Relevance in Inflammatory Bowel DiseaseImpact of psychological stress on irritable bowel syndromeReview article: the pathophysiological roles of the renin-angiotensin system in the gastrointestinal tractCentral muscarinic cholinergic activation alters interaction between splenic dendritic cell and CD4+CD25- T cells in experimental colitisSubstance P regulates migration in rat intestinal epithelial cellsEndothelin-converting enzyme-1 regulates trafficking and signalling of the neurokinin 1 receptor in endosomes of myenteric neuronesEndothelin-converting enzyme 1 promotes re-sensitization of neurokinin 1 receptor-dependent neurogenic inflammationNew function for an old enzyme: NEP deficient mice develop late-onset obesityStress and the Gastrointestinal Tract IV. Modulation of intestinal inflammation by stress: basic mechanisms and clinical relevance.Improved learning and memory in aged mice deficient in amyloid beta-degrading neutral endopeptidase.Endothelin-converting enzyme 1 and β-arrestins exert spatiotemporal control of substance P-induced inflammatory signals.Recent advances in inflammatory bowel disease.Adhesive small bowel obstruction: epidemiology, biology and prevention.Melanin-concentrating hormone (MCH) modulates C difficile toxin A-mediated enteritis in mice.Review article: mechanisms of initiation and perpetuation of gut inflammation by stress.Protective effects of neurokinin-1 receptor during colitis in mice: role of the epidermal growth factor receptor.Inflammation-induced abnormalities in the subcellular localization and trafficking of the neurokinin 1 receptor in the enteric nervous system.Endothelin-converting enzyme 1 degrades neuropeptides in endosomes to control receptor recycling.Irritable bowel syndrome: methods, mechanisms, and pathophysiology. Neural and neuro-immune mechanisms of visceral hypersensitivity in irritable bowel syndrome.Genetically altered animal models in the kallikrein-kinin system.Tachykinin receptors as drug targets for motility disorders.Impaired parasympathetic function increases susceptibility to inflammatory bowel disease in a mouse model of depressionImmunomodulatory properties of substance P: the gastrointestinal system as a model.Role of neuropeptides in inflammatory bowel disease.Interleukin-12 (IL-12) and IL-23 induction of substance p synthesis in murine T cells and macrophages is subject to IL-10 and transforming growth factor beta regulation.Neuroimmune interactions: potential target for mitigating or treating intestinal radiation injury.Contributions to the study of Trichinella spiralis TSL-1 antigens in host immunity.Dimethyl Fumarate Reduces Inflammatory Responses in Experimental Colitis.Neprilysin null mice develop exaggerated pulmonary vascular remodeling in response to chronic hypoxiaHCl-activated neural and epithelial vanilloid receptors (TRPV1) in cat esophageal mucosa.Tachykinins and their receptors: contributions to physiological control and the mechanisms of disease.Mechanisms underlying visceral hypersensitivity in irritable bowel syndrome.Arresting inflammation: contributions of plasma membrane and endosomal signalling to neuropeptide-driven inflammatory disease.Substance P and the regulation of inflammation in infections and inflammatory bowel disease.Neuropeptide substance P and the immune response.Long-term neprilysin inhibition - implications for ARNIs.Proliferation of prostate cancer cells and activity of neutral endopeptidase is regulated by bombesin and IL-1beta with IL-1beta acting as a modulator of cellular differentiation.Substance P-stimulated interleukin-8 expression in human colonic epithelial cells involves Rho family small GTPases.Protein kinase C-mediated desensitization of the neurokinin 1 receptor.
P2860
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P2860
Neutral endopeptidase (EC 3.4.24.11) terminates colitis by degrading substance P
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
1999年论文
@zh
1999年论文
@zh-cn
name
Neutral endopeptidase (EC 3.4.24.11) terminates colitis by degrading substance P
@ast
Neutral endopeptidase (EC 3.4.24.11) terminates colitis by degrading substance P
@en
type
label
Neutral endopeptidase (EC 3.4.24.11) terminates colitis by degrading substance P
@ast
Neutral endopeptidase (EC 3.4.24.11) terminates colitis by degrading substance P
@en
prefLabel
Neutral endopeptidase (EC 3.4.24.11) terminates colitis by degrading substance P
@ast
Neutral endopeptidase (EC 3.4.24.11) terminates colitis by degrading substance P
@en
P2093
P2860
P356
P1476
Neutral endopeptidase (EC 3.4.24.11) terminates colitis by degrading substance P
@en
P2093
N W Bunnett
P Geppetti
S M Collins
S Sturiale
P2860
P304
11653-11658
P356
10.1073/PNAS.96.20.11653
P407
P577
1999-09-01T00:00:00Z