Reduced ENaC protein abundance contributes to the lower blood pressure observed in pendrin-null mice. - Wikidata - awgldk - TriplyDB

Reduced ENaC protein abundance contributes to the lower blood pressure observed in pendrin-null mice.

Reduced ENaC protein abundance contributes to the lower blood pressure observed in pendrin-null mice.

http://www.wikidata.org/entity/Q50670408

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The epithelial anion transporter pendrin is induced by allergy and rhinovirus infection, regulates airway surface liquid, and increases airway reactivity and inflammation in an asthma model Collecting duct intercalated cell function and regulation α-Ketoglutarate regulates acid-base balance through an intrarenal paracrine mechanism Angiotensin II acts through the angiotensin 1a receptor to upregulate pendrin.Transcriptional regulation of the pendrin gene.Epithelial cell stretching and luminal acidification lead to a retarded development of stria vascularis and deafness in mice lacking pendrin.SLC26A4 gene polymorphism and late-onset Alzheimer's disease in a Han Chinese population from Qingdao, China.Pendrin modulates ENaC function by changing luminal HCO3-Regulation of two renal chloride transporters, AE1 and pendrin, by electrolytes and aldosterone The Cap1-claudin-4 regulatory pathway is important for renal chloride reabsorption and blood pressure regulation.IGF-1 and insulin exert opposite actions on ClC-K2 activity in the cortical collecting ducts Direct physical contact between intercalated cells in the distal convoluted tubule and the afferent arteriole in mouse kidneys.The V-ATPase B1-subunit promoter drives expression of Cre recombinase in intercalated cells of the kidney.Life-threatening metabolic alkalosis in Pendred syndrome Contractile force is enhanced in Aortas from pendrin null mice due to stimulation of angiotensin II-dependent signaling.Harvest and primary culture of the murine aldosterone-sensitive distal nephron Pendrin gene ablation alters ENaC subcellular distribution and open probability ENaC inhibition stimulates HCl secretion in the mouse cortical collecting duct. II. Bafilomycin-sensitive H+ secretion ENaC inhibition stimulates HCl secretion in the mouse cortical collecting duct. I. Stilbene-sensitive Cl- secretion.Pendrin localizes to the adrenal medulla and modulates catecholamine release A new look at electrolyte transport in the distal tubule ENaC inhibition stimulates Cl- secretion in the mouse cortical collecting duct through an NKCC1-dependent mechanism Pendrin protein abundance in the kidney is regulated by nitric oxide and cAMP Regulation of transport in the connecting tubule and cortical collecting duct The SLC26 gene family of anion transporters and channels.Maintaining K+ balance on the low-Na+, high-K+ diet.Nitric oxide reduces Cl⁻ absorption in the mouse cortical collecting duct through an ENaC-dependent mechanism.The role of pendrin in blood pressure regulation.Identification of allelic variants of pendrin (SLC26A4) with loss and gain of function.Renal β-intercalated cells maintain body fluid and electrolyte balance Novel diuretic targets Role of pendrin in iodide balance: going with the flow Developmental delays consistent with cochlear hypothyroidism contribute to failure to develop hearing in mice lacking Slc26a4/pendrin expression.Cortical distal nephron Cl(-) transport in volume homeostasis and blood pressure regulation.Regulation of extracellular fluid volume and blood pressure by pendrin.Blood pressure regulation via the epithelial sodium channel: from gene to kidney and beyond.Pendrin, a novel transcriptional target of the uroguanylin system.Electroneutral absorption of NaCl by the aldosterone-sensitive distal nephron: implication for normal electrolytes homeostasis and blood pressure regulation.Relative roles of principal and intercalated cells in the regulation of sodium balance and blood pressure.The enigma of continual plasma volume expansion in pregnancy: critical role of the renin-angiotensin-aldosterone system.

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P2860

Reduced ENaC protein abundance contributes to the lower blood pressure observed in pendrin-null mice.

http://www.wikidata.org/entity/Q50670408

description

2007 nî lūn-bûn

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2007年の論文

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年學術文章

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2007年學術文章

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name

Reduced ENaC protein abundance ...... observed in pendrin-null mice.

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Reduced ENaC protein abundance ...... observed in pendrin-null mice.

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type

label

Reduced ENaC protein abundance ...... observed in pendrin-null mice.

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Reduced ENaC protein abundance ...... observed in pendrin-null mice.

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prefLabel

Reduced ENaC protein abundance ...... observed in pendrin-null mice.

@en

Reduced ENaC protein abundance ...... observed in pendrin-null mice.

@nl

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American Journal of Physiology - Renal Physiology

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Reduced ENaC protein abundance ...... observed in pendrin-null mice.

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Jill W Verlander

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Kathryn B Spencer

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Lorraine A Everett

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Roy L Sutliff

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Susan M Wall

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Vladimir Pech

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William H Beierwaltes

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Wonkyong Shin

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Young Hee Kim

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P2860

Vasopressin increases water permeability of kidney collecting duct by inducing translocation of aquaporin-CD water channels to plasma membrane

Pendrin, encoded by the Pendred syndrome gene, resides in the apical region of renal intercalated cells and mediates bicarbonate secretion

Inhibition of prostasin-induced ENaC activities by PN-1 and regulation of PN-1 expression by TGF-beta1 and aldosterone

Intercalated cell H+/OH- transporter expression is reduced in Slc26a4 null mice

Long-term aldosterone treatment induces decreased apical but increased basolateral expression of AQP2 in CCD of rat kidney

Aquaporin-3 water channel localization and regulation in rat kidney

Regulated expression of pendrin in rat kidney in response to chronic NH4Cl or NaHCO3 loading.

Effects of six months of daily low-dose perchlorate exposure on thyroid function in healthy volunteers.

Aldosterone-mediated regulation of ENaC alpha, beta, and gamma subunit proteins in rat kidney.

Regulation of the abundance of renal sodium transporters and channels by vasopressin.

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