Advanced oxidation protein products promote NADPH oxidase-dependent β-cell destruction and dysfunction through the Bcl-2/Bax apoptotic pathway.

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Decreased diabetes risk over 9 year after 18-month oral L-arginine treatment in middle-aged subjects with impaired glucose tolerance and metabolic syndrome (extension evaluation of L-arginine study).

P2860

Q50101181-D3756E9D-2196-45EE-B8AC-18ECA03C4FF7

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Advanced oxidation protein products promote NADPH oxidase-dependent β-cell destruction and dysfunction through the Bcl-2/Bax apoptotic pathway.

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http://www.wikidata.org/entity/Q50860314

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年學術文章

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2017年學術文章

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name

Advanced oxidation protein pro ...... e Bcl-2/Bax apoptotic pathway.

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Advanced oxidation protein pro ...... e Bcl-2/Bax apoptotic pathway.

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Advanced oxidation protein pro ...... e Bcl-2/Bax apoptotic pathway.

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Advanced oxidation protein pro ...... e Bcl-2/Bax apoptotic pathway.

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Advanced oxidation protein pro ...... e Bcl-2/Bax apoptotic pathway.

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Advanced oxidation protein pro ...... e Bcl-2/Bax apoptotic pathway.

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P1476

Advanced oxidation protein pro ...... e Bcl-2/Bax apoptotic pathway.

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P2093

Aiju Lou

http://www.w3.org/2001/XMLSchema#string

Aiqing Li

http://www.w3.org/2001/XMLSchema#string

Fan Fan Hou

http://www.w3.org/2001/XMLSchema#string

Lei Yang

http://www.w3.org/2001/XMLSchema#string

Min Liang

http://www.w3.org/2001/XMLSchema#string

Shenglin Yang

http://www.w3.org/2001/XMLSchema#string

Xifang Zhang

http://www.w3.org/2001/XMLSchema#string

Youyuan Chen

http://www.w3.org/2001/XMLSchema#string

Yumin Li

http://www.w3.org/2001/XMLSchema#string

P2860

Epidemiologic and economic consequences of the global epidemics of obesity and diabetes

Salt-induced changes in cardiac phosphoproteome in a rat model of chronic renal failure.

High glucose inhibits glucose-6-phosphate dehydrogenase, leading to increased oxidative stress and beta-cell apoptosis.

Eicosanoids, β-cell function, and diabetes

Beta-cell glucose toxicity, lipotoxicity, and chronic oxidative stress in type 2 diabetes.

Angiotensin I-converting enzyme type 2 (ACE2) gene therapy improves glycemic control in diabetic mice.

Chronic oxidative stress as a central mechanism for glucose toxicity in pancreatic islet beta cells in diabetes.

Advanced oxidation protein products activate intrarenal renin-angiotensin system via a CD36-mediated, redox-dependent pathway.

Diabetes associated cell stress and dysfunction: role of mitochondrial and non-mitochondrial ROS production and activity.

NOX family NADPH oxidases in liver and in pancreatic islets: a role in the metabolic syndrome and diabetes?

P2888

labinvest.2017.24

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792-805

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scholarly article

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10.1038/LABINVEST.2017.24

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28263293

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apoptotic process

Advanced oxidation protein products promote NADPH oxidase-dependent β-cell destruction and dysfunction through the Bcl-2/Bax apoptotic pathway.

about

P2860

P2860

Advanced oxidation protein products promote NADPH oxidase-dependent β-cell destruction and dysfunction through the Bcl-2/Bax apoptotic pathway.

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