The DNA damage response (DDR) is induced by the C9orf72 repeat expansion in amyotrophic lateral sclerosis.
about
DNA strand breaks and TDP-43 mislocation are absent in the murine hSOD1G93A model of amyotrophic lateral sclerosis in vivo and in vitro.RNA Misprocessing in C9orf72-Linked Neurodegeneration.Rethinking Unconventional Translation in Neurodegeneration.Dysregulated molecular pathways in amyotrophic lateral sclerosis-frontotemporal dementia spectrum disorder.DNA plasticity and damage in amyotrophic lateral sclerosis.Perturbed autophagy and DNA repair converge to promote neurodegeneration in amyotrophic lateral sclerosis and dementia.Interactome analyses revealed that the U1 snRNP machinery overlaps extensively with the RNAP II machinery and contains multiple ALS/SMA-causative proteins.Searching the overlap between network modules with specific betweeness (S2B) and its application to cross-disease analysisNuclear poly(ADP-ribose) activity is a therapeutic target in amyotrophic lateral sclerosis
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The DNA damage response (DDR) is induced by the C9orf72 repeat expansion in amyotrophic lateral sclerosis.
description
2017 nî lūn-bûn
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name
The DNA damage response (DDR) ...... amyotrophic lateral sclerosis.
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The DNA damage response
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type
label
The DNA damage response (DDR) ...... amyotrophic lateral sclerosis.
@en
The DNA damage response
@nl
prefLabel
The DNA damage response (DDR) ...... amyotrophic lateral sclerosis.
@en
The DNA damage response
@nl
P2093
P2860
P356
P1476
The DNA damage response (DDR) ...... amyotrophic lateral sclerosis
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P2093
Daisuke Ito
Kai Ying Soo
Manal A Farg
P2860
P304
P356
10.1093/HMG/DDX170
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2017-08-01T00:00:00Z