Interferon γ is a STAT1-dependent direct inducer of BCL6 expression in imatinib-treated chronic myeloid leukemia cells.

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Inflammatory Signaling Pathways in Preleukemic and Leukemic Stem Cells.BCL6 as a therapeutic target for lymphoma.

P2860

Q47116205-2A48BD23-E765-4D1D-9D9C-3099FC282C51 Q47269833-A1CEF251-F2B6-459C-8755-F9A18C743B69

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Interferon γ is a STAT1-dependent direct inducer of BCL6 expression in imatinib-treated chronic myeloid leukemia cells.

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http://www.wikidata.org/entity/Q51075769

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年學術文章

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2017年學術文章

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name

Interferon γ is a STAT1-depend ...... hronic myeloid leukemia cells.

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Interferon γ is a STAT1-depend ...... hronic myeloid leukemia cells.

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type

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Interferon γ is a STAT1-depend ...... hronic myeloid leukemia cells.

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Interferon γ is a STAT1-depend ...... hronic myeloid leukemia cells.

@nl

prefLabel

Interferon γ is a STAT1-depend ...... hronic myeloid leukemia cells.

@en

Interferon γ is a STAT1-depend ...... hronic myeloid leukemia cells.

@nl

P1476

Interferon γ is a STAT1-depend ...... chronic myeloid leukemia cells

@en

P2093

D Salamon

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D Ujvari

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E Klein

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F Xue

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G Klein

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H Cheng

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H S Madapura

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J S McMurray

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L Stenke

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M Björkholm

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P2860

STAT5 represses BCL6 expression by binding to a regulatory region frequently mutated in lymphomas

Interferon-gamma: an overview of signals, mechanisms and functions

Interferons, immunity and cancer immunoediting

The BCL-2 protein family: opposing activities that mediate cell death

Cellular responses to interferon-gamma

A proto-oncogene BCL6 is up-regulated in the bone marrow microenvironment in multiple myeloma cells.

Isolation and killing of candidate chronic myeloid leukemia stem cells by antibody targeting of IL-1 receptor accessory protein.

The STAT5 inhibitor pimozide decreases survival of chronic myelogenous leukemia cells resistant to kinase inhibitors.

Transient expression of Bcl6 is sufficient for oncogenic function and induction of mature B-cell lymphoma.

Bim and Bad mediate imatinib-induced killing of Bcr/Abl+ leukemic cells, and resistance due to their loss is overcome by a BH3 mimetic.

P2888

onc.2017.85

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4619-4628

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scholarly article

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10.1038/ONC.2017.85

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English

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Marton Keszei

Lisa Westerberg

Sylvie Amu

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2017-04-03T00:00:00Z

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28368400

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P921

chronic myeloid leukemia

Interferon γ is a STAT1-dependent direct inducer of BCL6 expression in imatinib-treated chronic myeloid leukemia cells.

about

P2860

P2860

Interferon γ is a STAT1-dependent direct inducer of BCL6 expression in imatinib-treated chronic myeloid leukemia cells.

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