Neurites can remain viable after destruction of the neuronal soma by programmed cell death (apoptosis).
about
Endogenous Nmnat2 is an essential survival factor for maintenance of healthy axonsA local mechanism mediates NAD-dependent protection of axon degenerationThe progressive nature of Wallerian degeneration in wild-type and slow Wallerian degeneration (WldS) nervesStem cell autotomy and niche interaction in different systemsAxonal degeneration as a self-destructive defense mechanism against neurotropic virus infectionQuantitative and semi-quantitative measurements of axonal degeneration in tissue and primary neuron culturesWld(S) reduces paraquat-induced cytotoxicity via SIRT1 in non-neuronal cells by attenuating the depletion of NADAxon degeneration: Molecular mechanisms of a self-destruction pathwayGenetic influences on cellular reactions to brain injury: activation of microglia in denervated neuropil in mice carrying a mutation (Wld(S)) that causes delayed Wallerian degeneration.Clinical progression in Parkinson disease and the neurobiology of axons.Wallerian degeneration, wld(s), and nmnat.Unexpected survival of neurons of origin of the pyramidal tract after spinal cord injuryRetrograde and Wallerian axonal degeneration occur synchronously after retinal ganglion cell axotomy.Compartmental neurodegeneration and synaptic plasticity in the Wld(s) mutant mouse.Preserve and protect: maintaining axons within functional circuits.Sensory neuropathy attributable to loss of Bcl-w.Intracellular signalling pathways in dopamine cell death and axonal degeneration.Amyloid precursor protein cleavage-dependent and -independent axonal degeneration programs share a common nicotinamide mononucleotide adenylyltransferase 1-sensitive pathway.Axon pruning: an essential step underlying the developmental plasticity of neuronal connectionsNicotinamide mononucleotide adenylyl transferase 1 protects against acute neurodegeneration in developing CNS by inhibiting excitotoxic-necrotic cell death.A conserved function in phosphatidylinositol metabolism for mammalian Vps13 family proteinsAxonal degeneration is mediated by the mitochondrial permeability transition pore.gp130 cytokines are positive signals triggering changes in gene expression and axon outgrowth in peripheral neurons following injury.Elevated neuronal expression of CD200 protects Wlds mice from inflammation-mediated neurodegenerationDifferential proteomics analysis of synaptic proteins identifies potential cellular targets and protein mediators of synaptic neuroprotection conferred by the slow Wallerian degeneration (Wlds) gene.Protection of mouse retinal ganglion cell axons and soma from glaucomatous and ischemic injury by cytoplasmic overexpression of Nmnat1.UBE4B: a promising regulatory molecule in neuronal death and survival.Axons of retinal ganglion cells are insulted in the optic nerve early in DBA/2J glaucoma.Requirement of Neuronal Ribosome Synthesis for Growth and Maintenance of the Dendritic TreeModified cell cycle status in a mouse model of altered neuronal vulnerability (slow Wallerian degeneration; Wlds).The purine nucleosides adenosine and guanosine delay axonal degeneration in vitroNAD and axon degeneration: from the Wlds gene to neurochemistry.dSarm/Sarm1 is required for activation of an injury-induced axon death pathwayWld(S), Nmnats and axon degeneration--progress in the past two decades.Intrinsic axonal degeneration pathways are critical for glaucomatous damage.Axonal degeneration as a therapeutic target in the CNS.The ultimate question: why do axons degenerate? A tribute to the work and mentorship of John W. Griffin, MD.Wallerian degeneration: an emerging axon death pathway linking injury and disease.Mechanisms of developmental neurite pruning.Restoration versus reconstruction: cellular mechanisms of skin, nerve and muscle regeneration compared.
P2860
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P2860
Neurites can remain viable after destruction of the neuronal soma by programmed cell death (apoptosis).
description
1994 nî lūn-bûn
@nan
1994年の論文
@ja
1994年学术文章
@wuu
1994年学术文章
@zh
1994年学术文章
@zh-cn
1994年学术文章
@zh-hans
1994年学术文章
@zh-my
1994年学术文章
@zh-sg
1994年學術文章
@yue
1994年學術文章
@zh-hant
name
Neurites can remain viable aft ...... rammed cell death (apoptosis).
@en
Neurites can remain viable aft ...... soma by programmed cell death
@nl
type
label
Neurites can remain viable aft ...... rammed cell death (apoptosis).
@en
Neurites can remain viable aft ...... soma by programmed cell death
@nl
prefLabel
Neurites can remain viable aft ...... rammed cell death (apoptosis).
@en
Neurites can remain viable aft ...... soma by programmed cell death
@nl
P356
P1476
Neurites can remain viable aft ...... rammed cell death (apoptosis).
@en
P2093
Deckwerth TL
Johnson EM Jr
P356
10.1006/DBIO.1994.1234
P407
P577
1994-09-01T00:00:00Z