Oestrogen upregulates L-type Ca²⁺ channels via oestrogen-receptor- by a regional genomic mechanism in female rabbit hearts.
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Sex differences in the mechanisms underlying long QT syndromeBradycardia alters Ca(2+) dynamics enhancing dispersion of repolarization and arrhythmia riskDrug-induced long QT syndrome in women.Spectrum of Cav1.4 dysfunction in congenital stationary night blindness type 2A multiscale computational modelling approach predicts mechanisms of female sex risk in the setting of arousal-induced arrhythmias.MicroRNA-23a participates in estrogen deficiency induced gap junction remodeling of rats by targeting GJA1.How do sex hormones modify arrhythmogenesis in long QT syndrome? Sex hormone effects on arrhythmogenic substrate and triggered activity.Synchronous systolic subcellular Ca2+-elevations underlie ventricular arrhythmia in drug-induced long QT type 2.In silico Prediction of Sex-Based Differences in Human Susceptibility to Cardiac Ventricular TachyarrhythmiasThe link between abnormal calcium handling and electrical instability in acquired long QT syndrome--Does calcium precipitate arrhythmic storms?The female human heart at rest and during exercise: a review.Genomic upregulation of cardiac Cav1.2α and NCX1 by estrogen in women.Spontaneous initiation of premature ventricular complexes and arrhythmias in type 2 long QT syndrome.Molecular pathways of oestrogen receptors and β-adrenergic receptors in cardiac cells: Recognition of their similarities, interactions and therapeutic value.Sex and regional differences in rabbit right ventricular L-type calcium current levels and mathematical modelling of arrhythmia vulnerability.Activation of ClC-3 chloride channel by 17β-estradiol relies on the estrogen receptor α expression in breast cancer.Age-related differences in left ventricular structure and function between healthy men and women.Cross-sex hormonal replacement: is this really effective? an experimental clue.Another jigsaw piece in the complex picture of hormonal regulation of cardiac repolarization.Calcium in Brugada Syndrome: Questions for Future Research
P2860
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P2860
Oestrogen upregulates L-type Ca²⁺ channels via oestrogen-receptor- by a regional genomic mechanism in female rabbit hearts.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年学术文章
@wuu
2011年学术文章
@zh
2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
@yue
2011年學術文章
@zh-hant
name
Oestrogen upregulates L-type C ...... anism in female rabbit hearts.
@en
Oestrogen upregulates L-type C ...... anism in female rabbit hearts.
@nl
type
label
Oestrogen upregulates L-type C ...... anism in female rabbit hearts.
@en
Oestrogen upregulates L-type C ...... anism in female rabbit hearts.
@nl
prefLabel
Oestrogen upregulates L-type C ...... anism in female rabbit hearts.
@en
Oestrogen upregulates L-type C ...... anism in female rabbit hearts.
@nl
P2093
P2860
P1476
Oestrogen upregulates L-type C ...... anism in female rabbit hearts.
@en
P2093
Donald B Defranco
Fandian Zeng
Guy Salama
Xiaoyan Yang
P2860
P304
P356
10.1113/JPHYSIOL.2011.219501
P407
P50
P577
2011-11-28T00:00:00Z