Transgenic MSH overexpression attenuates the metabolic effects of a high-fat diet.
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Novel α-MSH analog causes weight loss in obese rats and minipigs and improves insulin sensitivityDeletion of prolyl carboxypeptidase attenuates the metabolic effects of diet-induced obesitySIRT1 deacetylase in POMC neurons is required for homeostatic defenses against diet-induced obesityEffects of AgRP inhibition on energy balance and metabolism in rodent modelsProlyl carboxypeptidase regulates energy expenditure and the thyroid axisEffects of a novel MC4R agonist on maintenance of reduced body weight in diet-induced obese mice.Nr4a1 siRNA expression attenuates α-MSH regulated gene expression in 3T3-L1 adipocytesPro-opiomelanocortin gene transfer to the nucleus of the solitary track but not arcuate nucleus ameliorates chronic diet-induced obesityInterleukin-18 null mutation increases weight and food intake and reduces energy expenditure and lipid substrate utilization in high-fat diet fed mice.Simultaneous POMC gene transfer to hypothalamus and brainstem increases physical activity, lipolysis and reduces adult-onset obesity.Hypothalamic proopiomelanocortin processing and the regulation of energy balanceThe melanocortin-4 receptor: physiology, pharmacology, and pathophysiology.β-Endorphin antagonizes the effects of α-MSH on food intake and body weightRegulation of prolactin in mice with altered hypothalamic melanocortin activity.Inflammation and neuropeptides: the connection in diabetic wound healing.Personalized nutrition and obesity.Temporal changes in nutritional state affect hypothalamic POMC peptide levels independently of leptin in adult male mice.Hypothalamic control of energy metabolism via the autonomic nervous system.Brain areas and pathways in the regulation of glucose metabolism.Participation of the central melanocortin system in metabolic regulation and energy homeostasis.IL-10 gene transfer upregulates arcuate POMC and ameliorates hyperphagia, obesity and diabetes by substituting for leptin.Intermittent MTII application evokes repeated anorexia and robust fat and weight loss.Lentivirus-mediated α-melanocyte-stimulating hormone overexpression in the hypothalamus decreases diet induced obesity in mice.Activation of the central melanocortin system chronically reduces body mass without the necessity of long-term caloric restriction.
P2860
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P2860
Transgenic MSH overexpression attenuates the metabolic effects of a high-fat diet.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh-hant
name
Transgenic MSH overexpression attenuates the metabolic effects of a high-fat diet.
@en
Transgenic MSH overexpression attenuates the metabolic effects of a high-fat diet.
@nl
type
label
Transgenic MSH overexpression attenuates the metabolic effects of a high-fat diet.
@en
Transgenic MSH overexpression attenuates the metabolic effects of a high-fat diet.
@nl
prefLabel
Transgenic MSH overexpression attenuates the metabolic effects of a high-fat diet.
@en
Transgenic MSH overexpression attenuates the metabolic effects of a high-fat diet.
@nl
P2093
P2860
P1476
Transgenic MSH overexpression attenuates the metabolic effects of a high-fat diet.
@en
P2093
Andrea Kim
Michelle Lee
Sharon L Wardlaw
Silvana Obici
Streamson C Chua
P2860
P304
P356
10.1152/AJPENDO.00555.2006
P577
2007-03-20T00:00:00Z