Quantitative ChIP-Seq normalization reveals global modulation of the epigenome.
about
Biological chromodynamics: a general method for measuring protein occupancy across the genome by calibrating ChIP-seq.Counteracting H3K4 methylation modulators Set1 and Jhd2 co-regulate chromatin dynamics and gene transcription.ZMYND8 Reads the Dual Histone Mark H3K4me1-H3K14ac to Antagonize the Expression of Metastasis-Linked GenesQuantitative analysis of ChIP-seq data uncovers dynamic and sustained H3K4me3 and H3K27me3 modulation in cancer cells under hypoxiaThe SWI/SNF chromatin remodelling complex is required for maintenance of lineage specific enhancersPotent and Selective KDM5 Inhibitor Stops Cellular Demethylation of H3K4me3 at Transcription Start Sites and Proliferation of MM1S Myeloma Cells.Epigenetic (re)programming of caste-specific behavior in the ant Camponotus floridanusRoles of H3K27me2 and H3K27me3 Examined during Fate Specification of Embryonic Stem CellsReference point insensitive molecular data analysis.Calibrating ChIP-Seq with Nucleosomal Internal Standards to Measure Histone Modification Density Genome Wide.Developmental Dynamics of X-Chromosome Dosage Compensation by the DCC and H4K20me1 in C. elegans.NEAT: a framework for building fully automated NGS pipelines and analyses.EpiMINE, a computational program for mining epigenomic data.An Alternative Approach to ChIP-Seq Normalization Enables Detection of Genome-Wide Changes in Histone H3 Lysine 27 Trimethylation upon EZH2 Inhibition.NucTools: analysis of chromatin feature occupancy profiles from high-throughput sequencing dataGenome-Wide Epigenetic Studies in Human Disease: A Primer on -Omic TechnologiesA Multiplexed System for Quantitative Comparisons of Chromatin Landscapes.Epigenomic profiling of primary gastric adenocarcinoma reveals super-enhancer heterogeneity.RYBP stimulates PRC1 to shape chromatin-based communication between Polycomb repressive complexes.An efficient targeted nuclease strategy for high-resolution mapping of DNA binding sites.HIST1H1C Regulates Interferon-β and Inhibits Influenza Virus Replication by Interacting with IRF3.The Overlooked Fact: Fundamental Need for Spike-In Control for Virtually All Genome-Wide AnalysesA novel SH2 recognition mechanism recruits Spt6 to the doubly phosphorylated RNA polymerase II linker at sites of transcription.A comprehensive comparison of tools for differential ChIP-seq analysis.Recent advances in ChIP-seq analysis: from quality management to whole-genome annotation.ChIP-seq in studying epigenetic mechanisms of disease and promoting precision medicine: progresses and future directions.Direct interrogation of the role of H3K9 in metazoan heterochromatin function.Reduced dosage of the chromosome axis factor Red1 selectively disrupts the meiotic recombination checkpoint in Saccharomyces cerevisiae.Histone H3K36 mutations promote sarcomagenesis through altered histone methylation landscape.Profiling Developmentally and Environmentally Controlled Chromatin Reprogramming.HDAC1,2 inhibition and doxorubicin impair Mre11-dependent DNA repair and DISC to override BCR-ABL1-driven DSB repair in Philadelphia chromosome-positive B-cell precursor acute lymphoblastic leukemia.---Inhibition of histone H3K27 demethylase selectively modulates inflammatory phenotypes of natural killer cells.H3.3K27M Cooperates with Trp53 Loss and PDGFRA Gain in Mouse Embryonic Neural Progenitor Cells to Induce Invasive High-Grade Gliomas.ChIP and ChIP-Related Techniques: Expanding the Fields of Application and Improving ChIP Performance.SETD2 alterations impair DNA damage recognition and lead to resistance to chemotherapy in leukemia.Pharmacological perturbation of CDK9 using selective CDK9 inhibition or degradation.Synthetic transcription elongation factors license transcription across repressive chromatin.DNA replication-coupled histone modification maintains Polycomb gene silencing in plants.Dot1 regulates nucleosome dynamics by its inherent histone chaperone activity in yeast.The Transcriptionally Permissive Chromatin State of Embryonic Stem Cells Is Acutely Tuned to Translational Output.
P2860
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P2860
Quantitative ChIP-Seq normalization reveals global modulation of the epigenome.
description
2014 nî lūn-bûn
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2014年の論文
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2014年学术文章
@wuu
2014年学术文章
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2014年学术文章
@zh-cn
2014年学术文章
@zh-hans
2014年学术文章
@zh-my
2014年学术文章
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2014年學術文章
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name
Quantitative ChIP-Seq normalization reveals global modulation of the epigenome.
@en
Quantitative ChIP-Seq normalization reveals global modulation of the epigenome.
@nl
type
label
Quantitative ChIP-Seq normalization reveals global modulation of the epigenome.
@en
Quantitative ChIP-Seq normalization reveals global modulation of the epigenome.
@nl
prefLabel
Quantitative ChIP-Seq normalization reveals global modulation of the epigenome.
@en
Quantitative ChIP-Seq normalization reveals global modulation of the epigenome.
@nl
P2093
P1433
P1476
Quantitative ChIP-Seq normalization reveals global modulation of the epigenome.
@en
P2093
Christian C Fritz
David A Orlando
Eric R Olson
James E Bradner
Matthew G Guenther
Mei Wei Chen
Snehakumari Solanki
Victoria E Brown
Yoon J Choi
P304
P356
10.1016/J.CELREP.2014.10.018
P577
2014-10-30T00:00:00Z