The ATG16L1 risk allele associated with Crohn's disease results in a Rac1-dependent defect in dendritic cell migration that is corrected by thiopurines.
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Interconnections between apoptotic and autophagic pathways during thiopurine-induced toxicity in cancer cells: the role of reactive oxygen species.Pathogenesis of Inflammatory Bowel Disease and Recent Advances in Biologic Therapies.Genetic control of autophagy underlies pathogenesis of inflammatory bowel disease.Trichothecenes: immunomodulatory effects, mechanisms, and anti-cancer potential.Modeling analysis of the relationship between atherosclerosis and related inflammatory factors.
P2860
The ATG16L1 risk allele associated with Crohn's disease results in a Rac1-dependent defect in dendritic cell migration that is corrected by thiopurines.
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2016 nî lūn-bûn
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2016年の論文
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2016年学术文章
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2016年学术文章
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2016年学术文章
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2016年学术文章
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2016年学术文章
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2016年學術文章
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The ATG16L1 risk allele associ ...... t is corrected by thiopurines.
@en
The ATG16L1 risk allele associ ...... t is corrected by thiopurines.
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type
label
The ATG16L1 risk allele associ ...... t is corrected by thiopurines.
@en
The ATG16L1 risk allele associ ...... t is corrected by thiopurines.
@nl
prefLabel
The ATG16L1 risk allele associ ...... t is corrected by thiopurines.
@en
The ATG16L1 risk allele associ ...... t is corrected by thiopurines.
@nl
P2093
P2860
P50
P356
P1433
P1476
The ATG16L1 risk allele associ ...... t is corrected by thiopurines.
@en
P2093
A A Te Velde
C J van der Woude
G R A M D'Haens
G R van den Brink
K Diederen
K I Anderson
M E Wildenberg
P J Koelink
S C S Wolfkamp
P2860
P2888
P304
P356
10.1038/MI.2016.65
P577
2016-07-20T00:00:00Z