Snail and slug play distinct roles during breast carcinoma progression.
about
Erk5 controls Slug expression and keratinocyte activation during wound healingThe epithelial-mesenchymal transition (EMT) phenomenonSustained activation of SMAD3/SMAD4 by FOXM1 promotes TGF-β-dependent cancer metastasisThe 'ins' and 'outs' of podosomes and invadopodia: characteristics, formation and functionEpithelial to Mesenchymal Transition in a Clinical PerspectiveTracking the intermediate stages of epithelial-mesenchymal transition in epithelial stem cells and cancerSLUG: Critical regulator of epithelial cell identity in breast development and cancerActivation of the canonical Wnt/β-catenin pathway in ATF3-induced mammary tumorsAdhesion in mammary development: novel roles for E-cadherin in individual and collective cell migrationDifferential role of Snail1 and Snail2 zinc fingers in E-cadherin repression and epithelial to mesenchymal transitionTwist1-induced dissemination preserves epithelial identity and requires E-cadherin.Characterization of the SNAG and SLUG domains of Snail2 in the repression of E-cadherin and EMT induction: modulation by serine 4 phosphorylationAn essential role for heat shock transcription factor binding protein 1 (HSBP1) during early embryonic developmentEpithelial-to-mesenchymal transition in pancreatic ductal adenocarcinoma: Characterization in a 3D-cell culture modelThe transcription factors Snail and Slug activate the transforming growth factor-beta signaling pathway in breast cancerSnail promotes CXCR2 ligand-dependent tumor progression in non-small cell lung carcinomaInduction of Slug by Chronic Exposure to Single-Walled Carbon Nanotubes Promotes Tumor Formation and Metastasis.Slug controls stem/progenitor cell growth dynamics during mammary gland morphogenesisStaurosporine augments EGF-mediated EMT in PMC42-LA cells through actin depolymerisation, focal contact size reduction and Snail1 induction - a model for cross-modulationExpression analysis of E-cadherin, Slug and GSK3beta in invasive ductal carcinoma of breast.SLUG/SNAI2 and tumor necrosis factor generate breast cells with CD44+/CD24- phenotype.Mode of action of the retrogene product SNAI1P, a SNAIL homolog, in human breast cancer cellsPhenotypic plasticity of neoplastic ovarian epithelium: unique cadherin profiles in tumor progressionSnail plays an oncogenic role in glioblastoma by promoting epithelial mesenchymal transition.Analysis of epithelial and mesenchymal markers in ovarian cancer reveals phenotypic heterogeneity and plasticity.Targeting signal transduction pathways to eliminate chemotherapeutic drug resistance and cancer stem cells.Clinicopathologic significance of slug expression in human intrahepatic cholangiocarcinoma.Stemness is Derived from Thyroid Cancer Cells.Differences in the transcriptional response to fulvestrant and estrogen deprivation in ER-positive breast cancerTranscription factors related to epithelial mesenchymal transition in tumor center and margin in invasive lung adenocarcinoma.Cyclin D1, Id1 and EMT in breast cancer.Key signalling nodes in mammary gland development and cancer. The Snail1-Twist1 conspiracy in malignant breast cancer progression.Protein kinase D1 maintains the epithelial phenotype by inducing a DNA-bound, inactive SNAI1 transcriptional repressor complex.Slug inhibits proliferation of human prostate cancer cells via downregulation of cyclin D1 expression.p66ShcA promotes breast cancer plasticity by inducing an epithelial-to-mesenchymal transition.The epithelial-mesenchymal transition (EMT) regulatory factor SLUG (SNAI2) is a downstream target of SPARC and AKT in promoting melanoma cell invasionCorrelation between Slug transcription factor and miR-221 in MDA-MB-231 breast cancer cells.Overexpression of Snail is associated with lymph node metastasis and poor prognosis in patients with gastric cancer.Lobular breast cancers lack the inverse relationship between ER/PR status and cell growth rate characteristic of ductal cancers in two independent patient cohorts: implications for tumor biology and adjuvant therapyBmi-1 promotes invasion and metastasis, and its elevated expression is correlated with an advanced stage of breast cancer
P2860
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P2860
Snail and slug play distinct roles during breast carcinoma progression.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
2006年學術文章
@zh-hant
name
Snail and slug play distinct roles during breast carcinoma progression.
@en
Snail and slug play distinct roles during breast carcinoma progression.
@nl
type
label
Snail and slug play distinct roles during breast carcinoma progression.
@en
Snail and slug play distinct roles during breast carcinoma progression.
@nl
prefLabel
Snail and slug play distinct roles during breast carcinoma progression.
@en
Snail and slug play distinct roles during breast carcinoma progression.
@nl
P2093
P50
P1476
Snail and slug play distinct roles during breast carcinoma progression
@en
P2093
Fabrice Magnino
Frédéric Bibeau
Pascal De Santa Barbara
P304
P356
10.1158/1078-0432.CCR-06-0478
P407
P577
2006-09-01T00:00:00Z