Different mechanisms of c-Jun NH(2)-terminal kinase-1 (JNK1) activation by ultraviolet-B radiation and by oxidative stressors.
about
Transcriptional profiling of MnSOD-mediated lifespan extension in Drosophila reveals a species-general network of aging and metabolic genesInfluence of light on aryl hydrocarbon receptor signaling and consequences in drug metabolism, physiology and diseaseD-MEKK1, the Drosophila orthologue of mammalian MEKK4/MTK1, and Hemipterous/D-MKK7 mediate the activation of D-JNK by cadmium and arsenite in Schneider cells.Ricin A-chain requires c-Jun N-terminal kinase to induce apoptosis in nontransformed epithelial cells.Administration of ricin induces a severe inflammatory response via nonredundant stimulation of ERK, JNK, and P38 MAPK and provides a mouse model of hemolytic uremic syndrome.The Anopheles gambiae oxidation resistance 1 (OXR1) gene regulates expression of enzymes that detoxify reactive oxygen speciesZAK is required for doxorubicin, a novel ribotoxic stressor, to induce SAPK activation and apoptosis in HaCaT cells.Intrapulmonary delivery of ricin at high dosage triggers a systemic inflammatory response and glomerular damage.Thioredoxin and thioredoxin target proteins: from molecular mechanisms to functional significance.Supreme EnLIGHTenment: damage recognition and signaling in the mammalian UV response.Skin fibroblasts from spermine synthase-deficient hemizygous gyro male (Gy/Y) mice overproduce spermidine and exhibit increased resistance to oxidative stress but decreased resistance to UV irradiation.The UV (Ribotoxic) stress response of human keratinocytes involves the unexpected uncoupling of the Ras-extracellular signal-regulated kinase signaling cascade from the activated epidermal growth factor receptor.c-Jun NH2-terminal kinase promotes apoptosis by down-regulating the transcriptional co-repressor CtBP.Retinoic acid and arsenic trioxide cooperate for apoptosis through phosphorylated RXR alpha.An apoptotic signaling pathway in the interferon antiviral response mediated by RNase L and c-Jun NH2-terminal kinase.Effects of heavy metals on mitogen-activated protein kinase pathways.Role of ATM in oxidative stress-mediated c-Jun phosphorylation in response to ionizing radiation and CdCl2.Amylin-induced cytotoxicity is associated with activation of caspase-3 and MAP kinases.Requirement for SAPK-JNK signaling in the induction of apoptosis by ribosomal stress in REH lymphoid leukemia cells.FoxO3a-mediated activation of stress responsive genes during early torpor in a mammalian hibernator.
P2860
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P2860
Different mechanisms of c-Jun NH(2)-terminal kinase-1 (JNK1) activation by ultraviolet-B radiation and by oxidative stressors.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
@yue
1999年學術文章
@zh-hant
name
Different mechanisms of c-Jun NH
@nl
Different mechanisms of c-Jun ...... on and by oxidative stressors.
@en
type
label
Different mechanisms of c-Jun NH
@nl
Different mechanisms of c-Jun ...... on and by oxidative stressors.
@en
prefLabel
Different mechanisms of c-Jun NH
@nl
Different mechanisms of c-Jun ...... on and by oxidative stressors.
@en
P2860
P356
P1476
Different mechanisms of c-Jun ...... on and by oxidative stressors.
@en
P2093
P2860
P304
25801-25806
P356
10.1074/JBC.274.36.25801
P407
P577
1999-09-01T00:00:00Z