Expression of Id1 results in apoptosis of cardiac myocytes through a redox-dependent mechanism.
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TGF-beta repression of Id2 induces apoptosis in gut epithelial cellsId-1, ITF-2, and Id-2 comprise a network of helix-loop-helix proteins that regulate mammary epithelial cell proliferation, differentiation, and apoptosis.Role of Id family proteins in growth control.Inhibition of Id1 augments insulin secretion and protects against high-fat diet-induced glucose intolerance.Expression profiling reveals distinct sets of genes altered during induction and regression of cardiac hypertrophyRegulatory functions of glutathione S-transferase P1-1 unrelated to detoxificationChanges of NF-kB, p53, Bcl-2 and caspase in apoptosis induced by JTE-522 in human gastric adenocarcinoma cell line AGS cells: role of reactive oxygen speciesDownregulation of Id1 by small interfering RNA in gastric cancer inhibits cell growth via the Akt pathway.Inflammatory properties of inhibitor of DNA binding 1 secreted by synovial fibroblasts in rheumatoid arthritis.Id1 overexpression is independent of repression and epigenetic silencing of tumor suppressor genes in melanomaStem cells, phenotypic inversion, and differentiation.BMP signaling dynamics in embryonic orofacial tissue.The impact of redox and thiol status on the bone marrow: Pharmacological intervention strategies.Id-1 gene downregulation by sulindac sulfide and its upregulation during tumor development in gastric cancer.The helix-loop-helix protein ID1 localizes to centrosomes and rapidly induces abnormal centrosome numbers.Microarray analysis uncovers retinoid targets in human bronchial epithelial cells.Id-1 expression promotes cell survival through activation of NF-kappaB signalling pathway in prostate cancer cells.ID1 enhances docetaxel cytotoxicity in prostate cancer cells through inhibition of p21.PPARgamma pathway activation results in apoptosis and COX-2 inhibition in HepG2 cellsMolecular cloning and characterization of a zinc finger protein involved in Id-1-stimulated mammary epithelial cell growth.The adenovirus E1A domains required for induction of DNA rereplication in G2/M arrested cells coincide with those required for apoptosis.Protein kinase C-delta modulates apoptosis induced by hyperglycemia in adult ventricular myocytes.Id2 expression during apoptosis and satellite cell activation in unloaded and loaded quail skeletal muscles.Denervation stimulates apoptosis but not Id2 expression in hindlimb muscles of aged rats.
P2860
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P2860
Expression of Id1 results in apoptosis of cardiac myocytes through a redox-dependent mechanism.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年学术文章
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1998年学术文章
@zh
1998年学术文章
@zh-cn
1998年学术文章
@zh-hans
1998年学术文章
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1998年学术文章
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1998年學術文章
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1998年學術文章
@zh-hant
name
Expression of Id1 results in a ...... h a redox-dependent mechanism.
@en
Expression of Id1 results in a ...... h a redox-dependent mechanism.
@nl
type
label
Expression of Id1 results in a ...... h a redox-dependent mechanism.
@en
Expression of Id1 results in a ...... h a redox-dependent mechanism.
@nl
prefLabel
Expression of Id1 results in a ...... h a redox-dependent mechanism.
@en
Expression of Id1 results in a ...... h a redox-dependent mechanism.
@nl
P2093
P2860
P356
P1476
Expression of Id1 results in a ...... h a redox-dependent mechanism.
@en
P2093
Deshpande SS
Ferrans VJ
Lowenstein CJ
P2860
P304
25922-25928
P356
10.1074/JBC.273.40.25922
P407
P577
1998-10-01T00:00:00Z