about
Strategies for the physiome project.Computational studies of the effects of myocardial blood flow reductions on cardiac metabolismThe cardiac muscle duplex as a method to study myocardial heterogeneityComputational model of erratic arrhythmias in a cardiac cell network: the role of gap junctionsUnraveling the genetics and mechanisms of cardiac arrhythmia.Model study of ATP and ADP buffering, transport of Ca(2+) and Mg(2+), and regulation of ion pumps in ventricular myocyteInhibitory effect of 2,3-butanedione monoxime (BDM) on Na(+)/Ca(2+) exchange current in guinea-pig cardiac ventricular myocytesCardiac models in drug discovery and development: a review.Modeling regulation of cardiac KATP and L-type Ca2+ currents by ATP, ADP, and Mg2+Ischemia reperfusion dysfunction changes model-estimated kinetics of myofilament interaction due to inotropic drugs in isolated hearts.A dynamic model of excitation-contraction coupling during acidosis in cardiac ventricular myocytes.Late sodium current in the pathophysiology of cardiovascular disease: consequences of sodium-calcium overload.Role of bicarbonate in the regulation of intracellular pH in the mammalian ventricular myocyte.Integrative modeling of the cardiac ventricular myocyte.Mitochondrial energetics, pH regulation, and ion dynamics: a computational-experimental approach.Modeling the heart.Acidosis in models of cardiac ventricular myocytes.Role of cellular compartmentation in the metabolic response to stress: mechanistic insights from computational models.Modeling cardiac ischemiaComputational approaches to understand cardiac electrophysiology and arrhythmias.Modeling cardiac action potential shortening driven by oxidative stress-induced mitochondrial oscillations in guinea pig cardiomyocytes.A model of Na+/H+ exchanger and its central role in regulation of pH and Na+ in cardiac myocytes.The modelling of a primitive 'sustainable' conservative cellNHE inhibition does not improve Na(+) or Ca(2+) overload during reperfusion: using modeling to illuminate the mechanisms underlying a therapeutic failure.Intrinsic H(+) ion mobility in the rabbit ventricular myocyte.Cross-bridge kinetics modeled from myoplasmic [Ca2+] and LV pressure at 17 degrees C and after 37 degrees C and 17 degrees C ischemia.Temperature dependence of Na+-H+ exchange, Na+-HCO3- co-transport, intracellular buffering and intracellular pH in guinea-pig ventricular myocytes.Effects of Na+/Ca2+ exchange induced by SR Ca2+ release on action potentials and afterdepolarizations in guinea pig ventricular myocytes.Mechanistic model of cardiac energy metabolism predicts localization of glycolysis to cytosolic subdomain during ischemia.Modeling transmural heterogeneity of K(ATP) current in rabbit ventricular myocytes.Characterization of intracellular pH regulation in the guinea-pig ventricular myocyte.Modeling oxygen requirements in ischemic cardiomyocytes.Ischemia-reperfusion injury changes the dynamics of Ca2+-contraction coupling due to inotropic drugs in isolated hearts.Regulation of cardiac energetics: role of redox state and cellular compartmentation during ischemia.
P2860
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P2860
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年学术文章
@wuu
1998年学术文章
@zh
1998年学术文章
@zh-cn
1998年学术文章
@zh-hans
1998年学术文章
@zh-my
1998年学术文章
@zh-sg
1998年學術文章
@yue
1998年學術文章
@zh-hant
name
Modelling myocardial ischaemia and reperfusion.
@en
Modelling myocardial ischaemia and reperfusion.
@nl
type
label
Modelling myocardial ischaemia and reperfusion.
@en
Modelling myocardial ischaemia and reperfusion.
@nl
prefLabel
Modelling myocardial ischaemia and reperfusion.
@en
Modelling myocardial ischaemia and reperfusion.
@nl
P2093
P1476
Modelling myocardial ischaemia and reperfusion.
@en
P2093
Vaughan-Jones RD
P304
P356
10.1016/S0079-6107(98)00023-6
P577
1998-01-01T00:00:00Z