Nitric oxide induces apoptosis via triggering mitochondrial permeability transition.
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Hepatitis C Virus Triggers Mitochondrial Permeability Transition with Production of Reactive Oxygen Species, Leading to DNA Damage and STAT3 ActivationProtective effect of cyclosporin A and FK506 from nitric oxide-dependent apoptosis in activated macrophagesEndogenous endothelial cell nitric-oxide synthase modulates apoptosis in cultured breast cancer cells and is transcriptionally regulated by p53S-Nitrosylation of mitochondrial caspasesIntracellular water motion decreases in apoptotic macrophages after caspase activation.Effect of solanine on the membrane potential of mitochondria in HepG2 cells and [Ca2+]i in the cells.Contrasting effects of NO and peroxynitrites on HSP70 expression and apoptosis in human monocytes.Inhibition of mitochondrial protein synthesis results in increased endothelial cell susceptibility to nitric oxide-induced apoptosisNitric oxide-induced genotoxicity, mitochondrial damage, and apoptosis in human lymphoblastoid cells expressing wild-type and mutant p53.Mechanisms of liver injury relevant to pediatric hepatology.The mitochondrial permeability transition pore regulates nitric oxide-mediated apoptosis of neurons induced by target deprivationNew advances on structural and biological functions of ceramide in apoptotic/necrotic cell death and cancer.Nitric oxide: a key regulator of myeloid inflammatory cell apoptosis.Adenovirus-mediated eNOS expression augments liver injury after ischemia/reperfusion in mice.The effect of nitric oxide on cell respiration: A key to understanding its role in cell survival or death.Thioredoxin reduces post-ischemic myocardial apoptosis by reducing oxidative/nitrative stress.Nitric oxide augments mesenchymal stem cell ability to repair liver fibrosis.Biochemical dysfunction in heart mitochondria exposed to ischaemia and reperfusion.Reactive oxygen species (ROS)-induced ROS release: a new phenomenon accompanying induction of the mitochondrial permeability transition in cardiac myocytes.The permeability transition pore complex: a target for apoptosis regulation by caspases and bcl-2-related proteins.Dynamics of mitochondrial structure during apoptosis and the enigma of Opa1Apoptosis induced by Rac GTPase correlates with induction of FasL and ceramides production.Methylsulfonylmethane modulates apoptosis of LPS/IFN-γ-activated RAW 264.7 macrophage-like cells by targeting p53, Bax, Bcl-2, cytochrome c and PARP proteins.Photoinduced nitric oxide release from a nitrobenzene derivative in mitochondria.Protection of cells from nitric oxide-mediated apoptotic death by glutathione C₆₀ derivative.Kaurane diterpenes protect against apoptosis and inhibition of phagocytosis in activated macrophages.Synergistic antiproliferative and apoptotic effects induced by mixed epidermal growth factor receptor inhibitor ZD1839 and nitric oxide donor in human prostatic cancer cell lines.Oxidative stress-induced apoptosis in retinal photoreceptor cells is mediated by calpains and caspases and blocked by the oxygen radical scavenger CR-6.Hypoxia sensitizes cells to nitric oxide-induced apoptosis.Microglial apoptosis induced by chromogranin A is mediated by mitochondrial depolarisation and the permeability transition but not by cytochrome c release.NO induces a cGMP-independent release of cytochrome c from mitochondria which precedes caspase 3 activation in insulin producing RINm5F cells.Tumor-initiating stem-like cells and drug resistance: carcinogenesis through Toll-like receptors, environmental factors, and virus.Nitric oxide-induced eosinophil apoptosis is dependent on mitochondrial permeability transition (mPT), JNK and oxidative stress: apoptosis is preceded but not mediated by early mPT-dependent JNK activation.Targeting Mitochondria by Olesoxime or Complement 1q Binding Protein as a Novel Management for Autism: A Hypothesis.Nitric oxide and its congeners in mitochondria: implications for apoptosis.Established Principles and Emerging Concepts on the Interplay between Mitochondrial Physiology and S-(De)nitrosylation: Implications in Cancer and Neurodegeneration.Resistance of activated human Th2 cells to NO-induced apoptosis is mediated by gamma-glutamyltranspeptidase.A transient inhibition of mitochondrial ATP synthesis by nitric oxide synthase activation triggered apoptosis in primary cortical neurons.Peroxisome proliferator-activated receptor-gamma-independent inhibition of macrophage activation by the non-thiazolidinedione agonist L-796,449. Comparison with the effects of 15-deoxy-delta(12,14)-prostaglandin J(2).The adenine nucleotide translocator: a target of nitric oxide, peroxynitrite, and 4-hydroxynonenal.
P2860
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P2860
Nitric oxide induces apoptosis via triggering mitochondrial permeability transition.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年学术文章
@wuu
1997年学术文章
@zh
1997年学术文章
@zh-cn
1997年学术文章
@zh-hans
1997年学术文章
@zh-my
1997年学术文章
@zh-sg
1997年學術文章
@yue
1997年學術文章
@zh-hant
name
Nitric oxide induces apoptosis via triggering mitochondrial permeability transition.
@en
Nitric oxide induces apoptosis via triggering mitochondrial permeability transition.
@nl
type
label
Nitric oxide induces apoptosis via triggering mitochondrial permeability transition.
@en
Nitric oxide induces apoptosis via triggering mitochondrial permeability transition.
@nl
prefLabel
Nitric oxide induces apoptosis via triggering mitochondrial permeability transition.
@en
Nitric oxide induces apoptosis via triggering mitochondrial permeability transition.
@nl
P2093
P50
P1433
P1476
Nitric oxide induces apoptosis via triggering mitochondrial permeability transition
@en
P2093
P304
P356
10.1016/S0014-5793(97)00623-6
P407
P577
1997-06-01T00:00:00Z