In cardiomyocyte hypoxia, insulin-like growth factor-I-induced antiapoptotic signaling requires phosphatidylinositol-3-OH-kinase-dependent and mitogen-activated protein kinase-dependent activation of the transcription factor cAMP response element-bi
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Apoptosis in myocardial ischaemia and infarctionWISP1, a pro-mitogenic, pro-survival factor, mediates tumor necrosis factor-alpha (TNF-alpha)-stimulated cardiac fibroblast proliferation but inhibits TNF-alpha-induced cardiomyocyte deathChronic mild hypoxia protects heart-derived H9c2 cells against acute hypoxia/reoxygenation by regulating expression of the SUR2A subunit of the ATP-sensitive K+ channelRegulation of cardiac autophagy by insulin-like growth factor 1Extracellular S100A1 protein inhibits apoptosis in ventricular cardiomyocytes via activation of the extracellular signal-regulated protein kinase 1/2 (ERK1/2)Regulation of phosphodiesterase 3 and inducible cAMP early repressor in the heartp53 initiates apoptosis by transcriptionally targeting the antiapoptotic protein ARCGlucose-dependent insulinotropic polypeptide-mediated up-regulation of beta-cell antiapoptotic Bcl-2 gene expression is coordinated by cyclic AMP (cAMP) response element binding protein (CREB) and cAMP-responsive CREB coactivator 2Growth hormone-releasing hormone promotes survival of cardiac myocytes in vitro and protects against ischaemia-reperfusion injury in rat heartMechanisms underlying hypoxia tolerance in Drosophila melanogaster: hairy as a metabolic switch.A brief review: adipose-derived stem cells and their therapeutic potential in cardiovascular diseases.PlGF repairs myocardial ischemia through mechanisms of angiogenesis, cardioprotection and recruitment of myo-angiogenic competent marrow progenitorsOverexpression of GD3 synthase induces apoptosis of vascular endothelial ECV304 cells through downregulation of Bcl-2.Simultaneous administration of insulin-like growth factor-1 and darbepoetin alfa protects the rat myocardium against myocardial infarction and enhances angiogenesisInvited Review: Autocrine/paracrine IGF-I and skeletal muscle adaptation.Effects of carvedilol treatment on cardiac cAMP response element binding protein expression and phosphorylation in acute coxsackievirus B3-induced myocarditisRole of the cyclic AMP response element in the bcl-2 promoter in the regulation of endogenous Bcl-2 expression and apoptosis in murine B cells.Corneal endothelial autocrine VIP enhances its integrity in stored human donor corneoscleral explant.Long-term methionine-diet induced mild hyperhomocysteinemia associated cardiac metabolic dysfunction in multiparous rats.Death begets failure in the heart.Distinctive ERK and p38 signaling in remote and infarcted myocardium during post-MI remodeling in the mouse.miR-29a promotes myocardial cell apoptosis induced by high glucose through down-regulating IGF-1Electrical and mechanical stimulation of cardiac cells and tissue constructs.Antiapoptotic effect of implanted embryonic stem cell-derived early-differentiated cells in aging rats after myocardial infarction.Inflammation and ischemia: macrophages activated by fibronectin fragments enhance the survival of injured cardiac myocytesProlyl hydroxylase domain protein 2 silencing enhances the survival and paracrine function of transplanted adipose-derived stem cells in infarcted myocardiumVIP down-regulates the inflammatory potential and promotes survival of dying (neural crest-derived) corneal endothelial cells ex vivo: necrosis to apoptosis switch and up-regulation of Bcl-2 and N-cadherinAkt mediated mitochondrial protection in the heart: metabolic and survival pathways to the rescue.Combination therapy with beta-adrenergic receptor antagonists and phosphodiesterase inhibitors for chronic heart failure.Protein-mediated Fatty Acid Uptake in the Heart.The cardioprotection of the insulin-mediated PI3K/Akt/mTOR signaling pathway.Application of adipose-derived stem cells in heart disease.Samul extract protects against the H2O2-induced apoptosis of H9c2 cardiomyoblasts via activation of extracellular regulated kinases (Erk) 1/2.Prolyl hydroxylase inhibitors act as agents to enhance the efficiency of cell therapy.CREB Negatively Regulates IGF2R Gene Expression and Downstream Pathways to Inhibit Hypoxia-Induced H9c2 Cardiomyoblast Cell Death.Protein kinase D links Gq-coupled receptors to cAMP response element-binding protein (CREB)-Ser133 phosphorylation in the heart.CREB participates in the IGF-I-stimulation cyclin D1 transcription.Inhibition of ROS-activated ERK1/2 pathway contributes to the protection of H2S against chemical hypoxia-induced injury in H9c2 cells.Restoration of CREB function ameliorates cisplatin cytotoxicity in renal tubular cells.Molecular Characterization and Expression Analysis of Insulin-like Growth Factor-1 and Insulin-like Growth Factor Binding Protein-1 Genes in Qinghai-Tibet Plateau Bos grunniens and Lowland Bos taurus
P2860
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P2860
In cardiomyocyte hypoxia, insulin-like growth factor-I-induced antiapoptotic signaling requires phosphatidylinositol-3-OH-kinase-dependent and mitogen-activated protein kinase-dependent activation of the transcription factor cAMP response element-bi
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
@zh
2001年学术文章
@zh-cn
2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
@yue
2001年學術文章
@zh-hant
name
In cardiomyocyte hypoxia, insu ...... actor cAMP response element-bi
@en
In cardiomyocyte hypoxia, insu ...... actor cAMP response element-bi
@nl
type
label
In cardiomyocyte hypoxia, insu ...... actor cAMP response element-bi
@en
In cardiomyocyte hypoxia, insu ...... actor cAMP response element-bi
@nl
prefLabel
In cardiomyocyte hypoxia, insu ...... actor cAMP response element-bi
@en
In cardiomyocyte hypoxia, insu ...... actor cAMP response element-bi
@nl
P2093
P356
P1433
P1476
In cardiomyocyte hypoxia, insu ...... actor cAMP response element-bi
@en
P2093
Bergmann MW
Mehrhof FB
von Harsdorf R
P304
P356
10.1161/HC4201.097133
P407
P577
2001-10-01T00:00:00Z