Morphologically normal-appearing mammary epithelial cells obtained from high-risk women exhibit methylation silencing of INK4a/ARF.
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Genomic Changes in Normal Breast Tissue in Women at Normal Risk or at High Risk for Breast CancerQuantitative methylation profiling in tumor and matched morphologically normal tissues from breast cancer patientsThe DNA damage-binding protein XPC is a frequent target for inactivation in squamous cell carcinomasBreast cancer genomes--form and functionSLIT/ROBO2 signaling promotes mammary stem cell senescence by inhibiting Wnt signaling.Comparison of Random Periareolar Fine Needle Aspirate versus Ductal Lavage for Risk Assessment and Prevention of Breast CancerCentrosome aberrations in human mammary epithelial cells driven by cooperative interactions between p16INK4a deficiency and telomere-dependent genotoxic stressReproducibility of random periareolar fine needle aspiration in a multi-institutional Cancer and Leukemia Group B (CALGB) cross-sectional study.Role of gene methylation in antitumor immune response: implication for tumor progression.CpG island tumor suppressor promoter methylation in non-BRCA-associated early mammary carcinogenesisStepwise DNA methylation changes are linked to escape from defined proliferation barriers and mammary epithelial cell immortalization.Familial and racial determinants of tumour suppressor genes promoter hypermethylation in breast tissues from healthy women.Silencing of the Lats2 tumor suppressor overrides a p53-dependent oncogenic stress checkpoint and enables mutant H-Ras-driven cell transformation.Epigenetic mechanisms in senescence, immortalisation and cancer.Tumor suppressor gene p16/INK4A/CDKN2A-dependent regulation into and out of the cell cycle in a spontaneous canine model of breast cancer.Gene Methylation and Cytological Atypia in Random Fine-Needle Aspirates for Assessment of Breast Cancer RiskDo we use the appropriate controls for the identification of informative methylation markers for early cancer detection?Mapping genomic and transcriptomic alterations spatially in epithelial cells adjacent to human breast carcinoma.Epigenetic Modifications as Biomarkers of Tumor Development, Therapy Response, and Recurrence across the Cancer Care Continuum.
P2860
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P2860
Morphologically normal-appearing mammary epithelial cells obtained from high-risk women exhibit methylation silencing of INK4a/ARF.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh-hant
name
Morphologically normal-appeari ...... lation silencing of INK4a/ARF.
@en
Morphologically normal-appeari ...... lation silencing of INK4a/ARF.
@nl
type
label
Morphologically normal-appeari ...... lation silencing of INK4a/ARF.
@en
Morphologically normal-appeari ...... lation silencing of INK4a/ARF.
@nl
prefLabel
Morphologically normal-appeari ...... lation silencing of INK4a/ARF.
@en
Morphologically normal-appeari ...... lation silencing of INK4a/ARF.
@nl
P2093
P1476
Morphologically normal-appeari ...... lation silencing of INK4a/ARF.
@en
P2093
Andrew D Bryson
Bruce F Kimler
Carol J Fabian
Carola M Zalles
Carolyn Paisie
Catherine Ibarra
Danielle M U Brander
Eric Dietze
Gregory R Bean
Joseph C Baker
P304
P356
10.1158/1078-0432.CCR-07-0407
P407
P433
P577
2007-11-01T00:00:00Z