COX-2 gene dosage-dependent defects in kidney development.
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Prostaglandin signaling regulates nephron segment patterning of renal progenitors during zebrafish kidney developmentAcetylsalicylic acid interferes with embryonic kidney growth and development by a prostaglandin-independent mechanism.Salt supplementation ameliorates developmental kidney defects in COX-2-/- mice.Differential compensation of two cyclooxygenases in renal homeostasis is independent of prostaglandin-synthetic capacity under basal conditions.
P2860
COX-2 gene dosage-dependent defects in kidney development.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年学术文章
@wuu
2016年学术文章
@zh-cn
2016年学术文章
@zh-hans
2016年学术文章
@zh-my
2016年学术文章
@zh-sg
2016年學術文章
@yue
2016年學術文章
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2016年學術文章
@zh-hant
name
COX-2 gene dosage-dependent defects in kidney development.
@en
COX-2 gene dosage-dependent defects in kidney development.
@nl
type
label
COX-2 gene dosage-dependent defects in kidney development.
@en
COX-2 gene dosage-dependent defects in kidney development.
@nl
prefLabel
COX-2 gene dosage-dependent defects in kidney development.
@en
COX-2 gene dosage-dependent defects in kidney development.
@nl
P2093
P2860
P1476
COX-2 gene dosage-dependent defects in kidney development.
@en
P2093
Patrick Slattery
Rolf M Nüsing
Stefanie Frölich
Yannik Schreiber
P2860
P304
P356
10.1152/AJPRENAL.00430.2015
P577
2016-03-16T00:00:00Z