Induction of neonatal sodium channel II and III alpha-isoform mRNAs in neurons and microglia after status epilepticus in the rat hippocampus.
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Trafficking mechanisms underlying neuronal voltage-gated ion channel localization at the axon initial segmentEvidence for a role of Nav1.6 in facilitating increases in neuronal hyperexcitability during epileptogenesis.Temporal lobe epilepsy induces intrinsic alterations in Na channel gating in layer II medial entorhinal cortex neurons.In situ-produced 7-chlorokynurenate has different effects on evoked responses in rats with limbic epilepsy in comparison to naive controls.Regulation of membrane excitability: a convergence on voltage-gated sodium conductance.Alternative splicing modulates inactivation of type 1 voltage-gated sodium channels by toggling an amino acid in the first S3-S4 linker.Nova2 interacts with a cis-acting polymorphism to influence the proportions of drug-responsive splice variants of SCN1A.The neurobiology of antiepileptic drugs for the treatment of nonepileptic conditions.Expression of voltage-gated sodium channel Nav1.3 is associated with severity of traumatic brain injury in adult ratsRapid loss of efficacy to the antiseizure drugs lamotrigine and carbamazepine: a novel experimental model of pharmacoresistant epilepsyEarly life stress as an influence on limbic epilepsy: an hypothesis whose time has come?Novel SCN3A variants associated with focal epilepsy in children.Properties of human brain sodium channel α-subunits expressed in HEK293 cells and their modulation by carbamazepine, phenytoin and lamotrigineNa+ channelopathies and epilepsy: recent advances and new perspectives.Carbamazepine and topiramate modulation of transient and persistent sodium currents studied in HEK293 cells expressing the Na(v)1.3 alpha-subunit.From Acupuncture to Interaction between δ-Opioid Receptors and Na (+) Channels: A Potential Pathway to Inhibit Epileptic Hyperexcitability.How Epilesy Changes Sodium ChannelsSeizure suppression through manipulating splicing of a voltage-gated sodium channelSodium channel beta1-subunit expression is increased in reactive astrocytes in a rat model for mesial temporal lobe epilepsy.Increased persistent sodium currents in rat entorhinal cortex layer V neurons in a post-status epilepticus model of temporal lobe epilepsy.Pro-excitatory alterations in sodium channel activity facilitate subiculum neuron hyperexcitability in temporal lobe epilepsy.Safinamide and flecainide protect axons and reduce microglial activation in models of multiple sclerosis.Kinetic changes and modulation by carbamazepine on voltage-gated sodium channels in rat CA1 neurons after epilepsy.Altered hippocampal gene expression prior to the onset of spontaneous seizures in the rat post-status epilepticus model.Intrinsic neuronal excitability: implications for health and disease.
P2860
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P2860
Induction of neonatal sodium channel II and III alpha-isoform mRNAs in neurons and microglia after status epilepticus in the rat hippocampus.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
@zh-cn
2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
@yue
2001年學術文章
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2001年學術文章
@zh-hant
name
Induction of neonatal sodium c ...... pticus in the rat hippocampus.
@en
Induction of neonatal sodium c ...... pticus in the rat hippocampus.
@nl
type
label
Induction of neonatal sodium c ...... pticus in the rat hippocampus.
@en
Induction of neonatal sodium c ...... pticus in the rat hippocampus.
@nl
prefLabel
Induction of neonatal sodium c ...... pticus in the rat hippocampus.
@en
Induction of neonatal sodium c ...... pticus in the rat hippocampus.
@nl
P2093
P2860
P1476
Induction of neonatal sodium c ...... pticus in the rat hippocampus.
@en
P2093
Lopes da Silva FH
van Vliet EA
P2860
P304
P356
10.1046/J.0953-816X.2001.01502.X
P407
P577
2001-03-01T00:00:00Z