Cardioprotective effect of diazoxide and its interaction with mitochondrial ATP-sensitive K+ channels. Possible mechanism of cardioprotection. - Wikidata - awgldk - TriplyDB

Cardioprotective effect of diazoxide and its interaction with mitochondrial ATP-sensitive K+ channels. Possible mechanism of cardioprotection.

Cardioprotective effect of diazoxide and its interaction with mitochondrial ATP-sensitive K+ channels. Possible mechanism of cardioprotection.

http://www.wikidata.org/entity/Q53962269

about

Multiprotein complex containing succinate dehydrogenase confers mitochondrial ATP-sensitive K+ channel activity Protein kinase Cepsilon interacts with and inhibits the permeability transition pore in cardiac mitochondria Immunolocalization of KATP channel subunits in mouse and rat cardiac myocytes and the coronary vasculature Pharmacological and ischemic preconditioning of the human myocardium: mitoK(ATP) channels are upstream and p38MAPK is downstream of PKC Reactive Oxygen Species, Endoplasmic Reticulum Stress and Mitochondrial Dysfunction: The Link with Cardiac Arrhythmogenesis Research progress of cardioprotective agents for prevention of anthracycline cardiotoxicity.Ion Channels and Oxidative Stress as a Potential Link for the Diagnosis or Treatment of Liver Diseases Mitochondrial ROMK channel is a molecular component of mitoK(ATP)Role of the MPTP in conditioning the heart - translatability and mechanism Targeting microglial K(ATP) channels to treat neurodegenerative diseases: a mitochondrial issue Physiological consequences of complex II inhibition for aging, disease, and the mKATP channel Mitochondrial K(ATP) channel activation reduces anoxic injury by restoring mitochondrial membrane potential Past and present course of cardioprotection against ischemia-reperfusion injury PKC-epsilon is upstream and PKC-alpha is downstream of mitoKATP channels in the signal transduction pathway of ischemic preconditioning of human myocardium Intramitochondrial signaling: interactions among mitoKATP, PKCepsilon, ROS, and MPT Investigation of mechanisms that mediate reactive hyperaemia in guinea-pig hearts: role of K(ATP) channels, adenosine, nitric oxide and prostaglandins The KATP channel opener diazoxide protects cardiac myocytes during metabolic inhibition without causing mitochondrial depolarization or flavoprotein oxidation Protein kinase C isoform-dependent modulation of ATP-sensitive K+ channels during reoxygenation in guinea-pig ventricular myocytes Diabetes mellitus reduces the function and expression of ATP-dependent K⁺ channels in cardiac mitochondria Potent cardioprotective effect of the 4-anilinoquinazoline derivative PD153035: involvement of mitochondrial K(ATP) channel activation Cardiac subsarcolemmal and interfibrillar mitochondria display distinct responsiveness to protection by diazoxide Magnesium Lithospermate B Protects Cardiomyocytes from Ischemic Injury Via Inhibition of TAB1-p38 Apoptosis Signaling Cardioprotective mechanism of diazoxide involves the inhibition of succinate dehydrogenase Mitochondrial adenine nucleotide transport and cardioprotection Sulfonylurea receptor ligands modulate stretch-induced ANF secretion in rat atrial myocyte culture.Physiology and pharmacology of myocardial preconditioning and postconditioning.Physiology and pharmacology of myocardial preconditioning.Reduction of ischemia and reperfusion-induced myocardial damage by cytochrome P450 inhibitors.Diazoxide triggers cardioprotection against apoptosis induced by oxidative stress.KATP channel openers: structure-activity relationships and therapeutic potential.Molecular identification and functional characterization of a mitochondrial sulfonylurea receptor 2 splice variant generated by intraexonic splicing.The mitochondrial complex II and ATP-sensitive potassium channel interaction: quantitation of the channel in heart mitochondria.Signal transduction in ischemic preconditioning: the role of kinases and mitochondrial K(ATP) channels.Polyphenol (-)-epigallocatechin gallate during ischemia limits infarct size via mitochondrial K(ATP) channel activation in isolated rat hearts Cellular mechanisms of infarct size reduction with ischemic preconditioning. Role of calcium?ATP-sensitive potassium currents from channels formed by Kir6 and a modified cardiac mitochondrial SUR2 variant Diazoxide protects against doxorubicin-induced cardiotoxicity in the rat.Diazoxide protects rat vascular endothelial cells against hypoxia and cold-induced damage.Abcc9 is required for the transition to oxidative metabolism in the newborn heart.Alternative mitochondrial functions in cell physiopathology: beyond ATP production.

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P2860

Cardioprotective effect of diazoxide and its interaction with mitochondrial ATP-sensitive K+ channels. Possible mechanism of cardioprotection.

http://www.wikidata.org/entity/Q53962269

description

1997 nî lūn-bûn

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1997年の論文

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1997年学术文章

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1997年学术文章

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1997年学术文章

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1997年学术文章

@zh-hans

1997年学术文章

@zh-my

1997年学术文章

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1997年學術文章

@yue

1997年學術文章

@zh-hant

name

Cardioprotective effect of dia ...... mechanism of cardioprotection.

@en

Cardioprotective effect of dia ...... mechanism of cardioprotection.

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type

label

Cardioprotective effect of dia ...... mechanism of cardioprotection.

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Cardioprotective effect of dia ...... mechanism of cardioprotection.

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prefLabel

Cardioprotective effect of dia ...... mechanism of cardioprotection.

@en

Cardioprotective effect of dia ...... mechanism of cardioprotection.

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01.RES.81.6.1072

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Circulation Research

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Cardioprotective effect of dia ...... mechanism of cardioprotection

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A J D'Alonzo

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G J Grover

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H N Murray

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M A Smith

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R B Darbenzio

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1072-1082

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scholarly article

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10.1161/01.RES.81.6.1072

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6

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81

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Vladimir Yarov-Yarovoy

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1997-12-01T00:00:00Z

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9400389

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