Activation of the PI3K/AKT pathway by microRNA-22 results in CLL B-cell proliferation.
about
MicroRNA regulation of lymphocyte tolerance and autoimmunityBRAF Mutation in Hairy Cell LeukemiamiR-125b and miR-532-3p predict the efficiency of rituximab-mediated lymphodepletion in chronic lymphocytic leukemia patients. A French Innovative Leukemia Organization study.The PU.1-Modulated MicroRNA-22 Is a Regulator of Monocyte/Macrophage Differentiation and Acute Myeloid Leukemia.Downregulated miR-495 [Corrected] Inhibits the G1-S Phase Transition by Targeting Bmi-1 in Breast Cancer.A comparison of microRNA expression profiles from splenic hemangiosarcoma, splenic nodular hyperplasia, and normal spleens of dogs.miR-141 is involved in BRD7-mediated cell proliferation and tumor formation through suppression of the PTEN/AKT pathway in nasopharyngeal carcinomaMolecular mechanisms and clinical applications of miR-22 in regulating malignant progression in human cancer (Review).Upregulation of miR-181c inhibits chemoresistance by targeting ST8SIA4 in chronic myelocytic leukemia.Factors involved in CLL pathogenesis and cell survival are disrupted by differentiation of CLL B-cells into antibody-secreting cells.The skewed balance between Tregs and Th17 in chronic lymphocytic leukemia.miR-330-3p controls cell proliferation by targeting early growth response 2 in non-small-cell lung cancer.Modeling the chronic lymphocytic leukemia microenvironment in vitro.Targeting protein kinase-b3 (akt3) signaling in melanoma.Tris (dibenzylideneacetone) dipalladium: a small-molecule palladium complex is effective in inducing apoptosis in chronic lymphocytic leukemia B-cells.Cell lines generated from a chronic lymphocytic leukemia mouse model exhibit constitutive Btk and Akt signaling.Ibrutinib downregulates a subset of miRNA leading to upregulation of tumor suppressors and inhibition of cell proliferation in chronic lymphocytic leukemia.Delineating the distinct role of AKT in mediating cell survival and proliferation induced by CD154 and IL-4/IL-21 in chronic lymphocytic leukemia.miR-22 in Smooth Muscle Cells: A Potential Therapy for Cardiovascular Disease.New Insights into Protein Kinase B/Akt Signaling: Role of Localized Akt Activation and Compartment-Specific Target Proteins for the Cellular Radiation Response.Biological Aspects of mTOR in Leukemia
P2860
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P2860
Activation of the PI3K/AKT pathway by microRNA-22 results in CLL B-cell proliferation.
description
2014 nî lūn-bûn
@nan
2014年の論文
@ja
2014年学术文章
@wuu
2014年学术文章
@zh-cn
2014年学术文章
@zh-hans
2014年学术文章
@zh-my
2014年学术文章
@zh-sg
2014年學術文章
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2014年學術文章
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2014年學術文章
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name
Activation of the PI3K/AKT pathway by microRNA-22 results in CLL B-cell proliferation.
@en
Activation of the PI3K/AKT pathway by microRNA-22 results in CLL B-cell proliferation.
@nl
type
label
Activation of the PI3K/AKT pathway by microRNA-22 results in CLL B-cell proliferation.
@en
Activation of the PI3K/AKT pathway by microRNA-22 results in CLL B-cell proliferation.
@nl
prefLabel
Activation of the PI3K/AKT pathway by microRNA-22 results in CLL B-cell proliferation.
@en
Activation of the PI3K/AKT pathway by microRNA-22 results in CLL B-cell proliferation.
@nl
P2093
P2860
P50
P356
P1433
P1476
Activation of the PI3K/AKT pathway by microRNA-22 results in CLL B-cell proliferation
@en
P2093
A I Landoni
F Palacios
G Dighiero
T Fernández-Calero
P2860
P2888
P304
P356
10.1038/LEU.2014.158
P577
2014-05-14T00:00:00Z