The hypothermic response to bacterial lipopolysaccharide critically depends on brain CB1, but not CB2 or TRPV1, receptors.
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Central neural regulation of brown adipose tissue thermogenesis and energy expenditureCold-Induced Thermogenesis and Inflammation-Associated Cold-Seeking Behavior Are Represented by Different Dorsomedial Hypothalamic Sites: A Three-Dimensional Functional Topography Study in Conscious RatsCentral nervous system regulation of brown adipose tissue.Monoacylglycerol Lipase Regulates Fever ResponseEndocannabinoid Catabolic Enzymes Play Differential Roles in Thermal Homeostasis in Response to Environmental or Immune Challenge.Brain CB₁ receptor expression following lipopolysaccharide-induced inflammation.Deletion of Monoglyceride Lipase in Astrocytes Attenuates Lipopolysaccharide-induced NeuroinflammationRapid and profound rewiring of brain lipid signaling networks by acute diacylglycerol lipase inhibition.Pharmacological blockade of the cold receptor TRPM8 attenuates autonomic and behavioral cold defenses and decreases deep body temperature.Transient receptor potential channel ankyrin-1 is not a cold sensor for autonomic thermoregulation in rodents.FAAH, but not MAGL, inhibition modulates acute TLR3-induced neuroimmune signaling in the rat, independent of sex.Obesity Impacts Fever and Sickness Behavior During Acute Systemic Inflammation.Intra-amniotic administration of lipopolysaccharide induces spontaneous preterm labor and birth in the absence of a body temperature change.Cannabinoid 1 receptors are critical for the innate immune response to TLR4 stimulation.Modulating sphingosine 1-phosphate signaling with DOP or FTY720 alleviates vascular and immune defects in mouse sepsis.Systemic administration of oleoylethanolamide protects from neuroinflammation and anhedonia induced by LPS in rats.Carbon monoxide down-modulates Toll-like receptor 4/MD2 expression on innate immune cells and reduces endotoxic shock susceptibility.TRPV1 antagonists that cause hypothermia, instead of hyperthermia, in rodents: compounds' pharmacological profiles, in-vivo targets, thermoeffectors recruited, and implications for drug development.
P2860
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P2860
The hypothermic response to bacterial lipopolysaccharide critically depends on brain CB1, but not CB2 or TRPV1, receptors.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年学术文章
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2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
@yue
2011年學術文章
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2011年學術文章
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name
The hypothermic response to ba ...... t not CB2 or TRPV1, receptors.
@en
The hypothermic response to ba ...... t not CB2 or TRPV1, receptors.
@nl
type
label
The hypothermic response to ba ...... t not CB2 or TRPV1, receptors.
@en
The hypothermic response to ba ...... t not CB2 or TRPV1, receptors.
@nl
prefLabel
The hypothermic response to ba ...... t not CB2 or TRPV1, receptors.
@en
The hypothermic response to ba ...... t not CB2 or TRPV1, receptors.
@nl
P2093
P2860
P50
P1476
The hypothermic response to ba ...... t not CB2 or TRPV1, receptors.
@en
P2093
Alla Y Molchanova
Andrej A Romanovsky
Daniela L Oliveira
Erika Pétervári
Justin Eales
M Devrim Dogan
Miklós Székely
Márta Balaskó
Narender R Gavva
Shreya Patel
P2860
P304
P356
10.1113/JPHYSIOL.2010.202465
P407
P577
2011-03-14T00:00:00Z