Expression of IL-31 gene transcripts in NC/Nga mice with atopic dermatitis.
about
New insights into the mechanisms of itch: are pain and itch controlled by distinct mechanisms?Roles of Mas-related G protein-coupled receptor X2 on mast cell-mediated host defense, pseudoallergic drug reactions, and chronic inflammatory diseasesIL-31-IL-31R interactions limit the magnitude of Th2 cytokine-dependent immunity and inflammation following intestinal helminth infectionItch mechanisms and circuits.Interleukin-31: a novel diagnostic marker of allergic diseases.Interleukin-31 pathway and its role in atopic dermatitis: a systematic review.Anti-IL-31 receptor antibody is shown to be a potential therapeutic option for treating itch and dermatitis in miceStructures and biological functions of IL-31 and IL-31 receptors.IL-31 Serum Protein and Tissue mRNA Levels in Patients with Atopic Dermatitis.Interleukin 31 mediates MAP kinase and STAT1/3 activation in intestinal epithelial cells and its expression is upregulated in inflammatory bowel disease.IL-31-IL-31R interactions negatively regulate type 2 inflammation in the lungProgressive activation of T(H)2/T(H)22 cytokines and selective epidermal proteins characterizes acute and chronic atopic dermatitis.Analytic review: Interleukin-6 in surgery, trauma, and critical care: part I: basic science.Medical treatment of pruritus.Investigational drugs for pruritus.Regulation of Th2 responses by different cell types expressing the interleukin-31 receptorPreclinical and human surrogate models of itch.Transcriptional activation of the IL31 gene by NFAT and STAT6.Definition and characterization of an inhibitor for interleukin-31.Chronic pruritus: clinics and treatmentMolecular dissection of human interleukin-31-mediated signal transduction through site-directed mutagenesis.Increased scratching counts depend on a decrease in ability of cutaneous prostaglandin D2 biosynthesis in NC/Nga mice with atopic dermatitis.Involvement of IL-31 on scratching behavior in NC/Nga mice with atopic-like dermatitis.IL-1β induces thymic stromal lymphopoietin and an atopic dermatitis-like phenotype in reconstructed healthy human epidermis.IL-31 significantly correlates with disease activity and Th2 cytokine levels in children with atopic dermatitis.Human basophils are a source of - and are differentially activated by - IL-31.IL-31 is crucial for induction of pruritus, but not inflammation, in contact hypersensitivity.Diagnosis and treatment of atopic dermatitis in children and adults: European Academy of Allergology and Clinical Immunology/American Academy of Allergy, Asthma and Immunology/PRACTALL Consensus Report
P2860
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P2860
Expression of IL-31 gene transcripts in NC/Nga mice with atopic dermatitis.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
Expression of IL-31 gene transcripts in NC/Nga mice with atopic dermatitis.
@en
Expression of IL-31 gene transcripts in NC/Nga mice with atopic dermatitis.
@nl
type
label
Expression of IL-31 gene transcripts in NC/Nga mice with atopic dermatitis.
@en
Expression of IL-31 gene transcripts in NC/Nga mice with atopic dermatitis.
@nl
prefLabel
Expression of IL-31 gene transcripts in NC/Nga mice with atopic dermatitis.
@en
Expression of IL-31 gene transcripts in NC/Nga mice with atopic dermatitis.
@nl
P2093
P1476
Expression of IL-31 gene transcripts in NC/Nga mice with atopic dermatitis.
@en
P2093
Akie Yamaguchi
Akiko Takaoka
Makoto Tanaka
Masanori Sugimoto
Shiro Nakaike
P304
P356
10.1016/J.EJPHAR.2005.04.040
P407
P577
2005-06-01T00:00:00Z