MLR-1023 is a potent and selective allosteric activator of Lyn kinase in vitro that improves glucose tolerance in vivo.
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Src family kinases differentially influence glioma growth and motilityNovel approaches for targeting kinases: allosteric inhibition, allosteric activation and pseudokinases.-NOD Mice Having a Lyn Tyrosine Kinase Mutation Exhibit Abnormal Neutrophil Chemotaxis.Cholesterol efflux in megakaryocyte progenitors suppresses platelet production and thrombocytosis.Integrin CD11b negatively regulates Mincle-induced signaling via the Lyn-SIRPα-SHP1 complex.
P2860
MLR-1023 is a potent and selective allosteric activator of Lyn kinase in vitro that improves glucose tolerance in vivo.
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2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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MLR-1023 is a potent and selec ...... ves glucose tolerance in vivo.
@en
MLR-1023 is a potent and selec ...... ves glucose tolerance in vivo.
@nl
type
label
MLR-1023 is a potent and selec ...... ves glucose tolerance in vivo.
@en
MLR-1023 is a potent and selec ...... ves glucose tolerance in vivo.
@nl
prefLabel
MLR-1023 is a potent and selec ...... ves glucose tolerance in vivo.
@en
MLR-1023 is a potent and selec ...... ves glucose tolerance in vivo.
@nl
P2093
P356
P1476
MLR-1023 is a potent and selec ...... ves glucose tolerance in vivo.
@en
P2093
Alexander R Ochman
Andrew G Reaume
Jeffrey A Handler
P356
10.1124/JPET.112.192096
P407
P577
2012-04-03T00:00:00Z