Poly(ADP-ribose) polymerase 1 activation is required for cisplatin nephrotoxicity.
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Programmed necrosis in acute kidney injuryA Role for Tubular Necroptosis in Cisplatin-Induced AKIRegulation of necrotic cell death: p53, PARP1 and cyclophilin D-overlapping pathways of regulated necrosis?Pathophysiology of cisplatin-induced acute kidney injury.Inhibition of soluble epoxide hydrolase prevents renal interstitial fibrosis and inflammation.Targeted deletion of p53 in the proximal tubule prevents ischemic renal injuryRegulated cell death in AKI.Enhanced susceptibility of ovaries from obese mice to 7,12-dimethylbenz[a]anthracene-induced DNA damageTIGAR regulates glycolysis in ischemic kidney proximal tubules.Pharmacological inhibition of soluble epoxide hydrolase prevents renal interstitial fibrogenesis in obstructive nephropathy.Cisplatin induces primary necrosis through poly(ADP-ribose) polymerase 1 activation in kidney proximal tubular cellsRenal nerves drive interstitial fibrogenesis in obstructive nephropathy.Poly(ADP-ribose) polymerase regulates glycolytic activity in kidney proximal tubule epithelial cellsRole of CFTR in oxidative stress and suicidal death of renal cells during cisplatin-induced nephrotoxicity.Poly(ADP-ribose) polymerase 1 contributes to oxidative stress through downregulation of sirtuin 3 during cisplatin nephrotoxicityParp1 protects against Aag-dependent alkylation-induced nephrotoxicity in a sex-dependent mannerRole of poly (ADP-ribose)-polymerase and its signaling pathway with renin-angiotensin aldosterone system in renal diseases.Necroinflammation in Kidney Disease.Apoptosis inhibitor of macrophage protein enhances intraluminal debris clearance and ameliorates acute kidney injury in mice.β-Lapachone ameliorates murine cisplatin nephrotoxicity: NAD⁺, NQO1, and SIRT1 at the crossroads of metabolism, injury, and inflammationSpermidine is protective against kidney ischemia and reperfusion injury through inhibiting DNA nitration and PARP1 activation.Panax notoginseng saponins mitigate cisplatin induced nephrotoxicity by inducing mitophagy via HIF-1α.Spermidine rescues proximal tubular cells from oxidative stress and necrosis after ischemic acute kidney injury.Olaparib protects cardiomyocytes against oxidative stress and improves graft contractility during the early phase after heart transplantation in rats.Heme Oxygenase-1 Mitigates Ferroptosis in Renal Proximal Tubule Cells.Inhibition of PARP activation by enalapril is crucial for its renoprotective effect in cisplatin-induced nephrotoxicity in rats.The protective effect of trimetazidine against cisplatin-induced nephrotoxicity in rats.Cisplatin-induced oxidative stress stimulates renal Fas ligand shedding.5-aminoisoquinoline improves renal function and fibrosis during recovery phase of cisplatin-induced acute kidney injury in rats.Exogenous spermidine ameliorates tubular necrosis during cisplatin nephrotoxicity
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P2860
Poly(ADP-ribose) polymerase 1 activation is required for cisplatin nephrotoxicity.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
@zh
2012年學術文章
@zh-hant
name
Poly(ADP-ribose) polymerase 1 activation is required for cisplatin nephrotoxicity.
@en
type
label
Poly(ADP-ribose) polymerase 1 activation is required for cisplatin nephrotoxicity.
@en
prefLabel
Poly(ADP-ribose) polymerase 1 activation is required for cisplatin nephrotoxicity.
@en
P2093
P356
P1433
P1476
Poly(ADP-ribose) polymerase 1 activation is required for cisplatin nephrotoxicity.
@en
P2093
Babu J Padanilam
Kelly E Long
P2888
P304
P356
10.1038/KI.2012.64
P407
P577
2012-03-21T00:00:00Z
P5875
P6179
1001479627