Nuclear factor kappaB downregulates the transient outward potassium current I(to,f) through control of KChIP2 expression.
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Long QT Syndrome: An Emerging Role for Inflammation and ImmunitySubclinical Detection of Diabetic Cardiomyopathy with MicroRNAs: Challenges and PerspectivesTGF-β1, released by myofibroblasts, differentially regulates transcription and function of sodium and potassium channels in adult rat ventricular myocytesMicroRNA-301a mediated regulation of Kv4.2 in diabetes: identification of key modulators.Improvement of cardiac contractile function by peptide-based inhibition of NF-κB in the utrophin/dystrophin-deficient murine model of muscular dystrophy.Simvastatin modulates remodeling of Kv4.3 expression in rat hypertrophied cardiomyocytes.Different KChIPs compete for heteromultimeric assembly with pore-forming Kv4 subunitsEffects of C-reactive protein on K(+) channel interaction protein 2 in cardiomyocytes.Impact of KChIP2 on Cardiac Electrophysiology and the Progression of Heart Failure.Mechanisms of ventricular arrhythmias: a dynamical systems-based perspective.Exercise training and PI3Kα-induced electrical remodeling is independent of cellular hypertrophy and Akt signalingReductions in the Cardiac Transient Outward K+ Current Ito Caused by Chronic β-Adrenergic Receptor Stimulation Are Partly Rescued by Inhibition of Nuclear Factor κB.Auxiliary KChIP4a suppresses A-type K+ current through endoplasmic reticulum (ER) retention and promoting closed-state inactivation of Kv4 channelsHyperoxia-induced hypertrophy and ion channel remodeling in left ventricleMarked QTc Prolongation and Torsades de pointes in Patients with Chronic Inflammatory Arthritis.KChIP2 is a core transcriptional regulator of cardiac excitability.Cytokine Effects on Mechano-Induced Electrical Activity in Atrial Myocardium.Extracellular signal-regulated kinase activation during cardiac hypertrophy reduces sarcoplasmic/endoplasmic reticulum calcium ATPase 2 (SERCA2) transcription.Molecular Mechanisms and New Treatment Paradigm for Atrial Fibrillation.Antiarrhythmic potential of anticytokine therapy in rheumatoid arthritis: tocilizumab reduces corrected QT interval by controlling systemic inflammation.Elevated potassium outward currents in hyperoxia treated atrial cardiomyocytes.
P2860
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P2860
Nuclear factor kappaB downregulates the transient outward potassium current I(to,f) through control of KChIP2 expression.
description
2011 nî lūn-bûn
@nan
2011年の論文
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2011年学术文章
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2011年学术文章
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2011年学术文章
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2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
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2011年學術文章
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2011年學術文章
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name
Nuclear factor kappaB downregu ...... control of KChIP2 expression.
@en
Nuclear factor kappaB downregu ...... control of KChIP2 expression.
@nl
type
label
Nuclear factor kappaB downregu ...... control of KChIP2 expression.
@en
Nuclear factor kappaB downregu ...... control of KChIP2 expression.
@nl
prefLabel
Nuclear factor kappaB downregu ...... control of KChIP2 expression.
@en
Nuclear factor kappaB downregu ...... control of KChIP2 expression.
@nl
P2093
P1433
P1476
Nuclear factor kappaB downregu ...... control of KChIP2 expression.
@en
P2093
Brian K Panama
Desiree Latour-Villamil
Dongling Zhao
Gerrie P Farman
Lorrie A Kirshenbaum
Peter H Backx
Steffen-Sebastian Bolz
P304
P356
10.1161/CIRCRESAHA.110.229112
P577
2011-01-20T00:00:00Z