Prucalopride inhibits the glioma cells proliferation and induces autophagy via AKT-mTOR pathway.

about

Prucalopride inhibits the glioma cells proliferation and induces autophagy via AKT-mTOR pathway.

Item

http://www.wikidata.org/entity/Q54977129

description

2018 nî lūn-bûn

@nan

2018年の論文

@ja

2018年学术文章

@wuu

2018年学术文章

@zh-cn

2018年学术文章

@zh-hans

2018年学术文章

@zh-my

2018年学术文章

@zh-sg

2018年學術文章

@yue

2018年學術文章

@zh

2018年學術文章

@zh-hant

name

Prucalopride inhibits the glio ...... utophagy via AKT-mTOR pathway.

@en

Prucalopride inhibits the glio ...... utophagy via AKT-mTOR pathway.

@nl

type

Item

label

Prucalopride inhibits the glio ...... utophagy via AKT-mTOR pathway.

@en

Prucalopride inhibits the glio ...... utophagy via AKT-mTOR pathway.

@nl

prefLabel

Prucalopride inhibits the glio ...... utophagy via AKT-mTOR pathway.

@en

Prucalopride inhibits the glio ...... utophagy via AKT-mTOR pathway.

@nl

P1476

Prucalopride inhibits the glio ...... utophagy via AKT-mTOR pathway.

@en

P2093

Hong Qiao

http://www.w3.org/2001/XMLSchema#string

Li-Li Bi

http://www.w3.org/2001/XMLSchema#string

Yong-Bo Wang

http://www.w3.org/2001/XMLSchema#string

Yu-Mei Gao

http://www.w3.org/2001/XMLSchema#string

P2860

Epigenetic pathways and glioblastoma treatment

Expression of serotonin receptors 2B and 4 in human prostate cancer tissue and effects of their antagonists on prostate cancer cell lines

Activation of 5-HT4 receptors facilitates neurogenesis of injured enteric neurons at an anastomosis in the lower gut

Autophagy in the Pathogenesis of Disease

Malignant gliomas in adults

Autophagy and metabolism

5-HT4 receptor-mediated neuroprotection and neurogenesis in the enteric nervous system of adult mice.

Deciphering downstream gene targets of PI3K/mTOR/p70S6K pathway in breast cancer

Itraconazole suppresses the growth of glioblastoma through induction of autophagy: involvement of abnormal cholesterol trafficking.

The brain 5-HT4 receptor binding is down-regulated in the Flinders Sensitive Line depression model and in response to paroxetine administration.

P2888

s12883-018-1083-7

P304

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1186/S12883-018-1083-7

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2018-06-04T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P6179

1104385385

http://www.w3.org/2001/XMLSchema#string

P698

29866060

http://www.w3.org/2001/XMLSchema#string

P932

5985575

http://www.w3.org/2001/XMLSchema#string