Alzheimer's Transgenic Model Is Characterized by Very Early Brain Network Alterations and β-CTF Fragment Accumulation: Reversal by β-Secretase Inhibition.

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Alzheimer's Transgenic Model Is Characterized by Very Early Brain Network Alterations and β-CTF Fragment Accumulation: Reversal by β-Secretase Inhibition.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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2018年學術文章

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Alzheimer's Transgenic Model I ...... sal by β-Secretase Inhibition.

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Alzheimer's Transgenic Model I ...... sal by β-Secretase Inhibition.

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type

Item

label

Alzheimer's Transgenic Model I ...... sal by β-Secretase Inhibition.

@en

Alzheimer's Transgenic Model I ...... sal by β-Secretase Inhibition.

@nl

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Alzheimer's Transgenic Model I ...... sal by β-Secretase Inhibition.

@en

Alzheimer's Transgenic Model I ...... sal by β-Secretase Inhibition.

@nl

P1476

Alzheimer's Transgenic Model I ...... sal by β-Secretase Inhibition.

@en

P2093

Frederic Manseau

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Ning Gu

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Siddhartha Mondragón-Rodríguez

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Sylvain Williams

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P2860

Inhibitory interneuron deficit links altered network activity and cognitive dysfunction in Alzheimer model.

The subiculum: what it does, what it might do, and what neuroanatomy has yet to tell us

Discovery of AZD3839, a Potent and Selective BACE1 Inhibitor Clinical Candidate for the Treatment of Alzheimer Disease

High-level neuronal expression of abeta 1-42 in wild-type human amyloid protein precursor transgenic mice: synaptotoxicity without plaque formation

High gamma power is phase-locked to theta oscillations in human neocortex

Increased EEG gamma band activity in Alzheimer's disease and mild cognitive impairment.

Many neuronal and behavioral impairments in transgenic mouse models of Alzheimer's disease are independent of caspase cleavage of the amyloid precursor protein

Parvalbumin-expressing interneurons coordinate hippocampal network dynamics required for memory consolidation.

Desynchronization of neocortical networks by asynchronous release of GABA at autaptic and synaptic contacts from fast-spiking interneurons.

Place cells, grid cells, and the brain's spatial representation system.

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121

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scholarly article

P356

10.3389/FNCEL.2018.00121

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2018-05-08T00:00:00Z

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29867356

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5952042

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