Heterozygous CDKL5 Knockout Female Mice Are a Valuable Animal Model for CDKL5 Disorder.

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Heterozygous CDKL5 Knockout Female Mice Are a Valuable Animal Model for CDKL5 Disorder.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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2018年學術文章

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2018年學術文章

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Heterozygous CDKL5 Knockout Female Mice Are a Valuable Animal Model for CDKL5 Disorder.

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Heterozygous CDKL5 Knockout Female Mice Are a Valuable Animal Model for CDKL5 Disorder.

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Heterozygous CDKL5 Knockout Female Mice Are a Valuable Animal Model for CDKL5 Disorder.

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Heterozygous CDKL5 Knockout Female Mice Are a Valuable Animal Model for CDKL5 Disorder.

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Heterozygous CDKL5 Knockout Female Mice Are a Valuable Animal Model for CDKL5 Disorder

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Heterozygous CDKL5 Knockout Female Mice Are a Valuable Animal Model for CDKL5 Disorder.

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Heterozygous CDKL5 Knockout Female Mice Are a Valuable Animal Model for CDKL5 Disorder.

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P1476

Heterozygous CDKL5 Knockout Female Mice Are a Valuable Animal Model for CDKL5 Disorder

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P2093

Claudia Fuchs

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Elisa Ren

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Giovanna Zoccoli

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Roberto Rimondini

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Simone Bettini

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Stefania Trazzi

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Stefano Bastianini

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Viviana Lo Martire

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P2860

Protein measurement with the Folin phenol reagent

Functional consequences of mutations in CDKL5, an X-linked gene involved in infantile spasms and mental retardation

CDKL5 belongs to the same molecular pathway of MeCP2 and it is responsible for the early-onset seizure variant of Rett syndrome

Identification and characterization of a novel serine-threonine kinase gene from the Xp22 region

Disruption of the serine/threonine kinase 9 gene causes severe X-linked infantile spasms and mental retardation

CDKL5 expression is modulated during neuronal development and its subcellular distribution is tightly regulated by the C-terminal tail

Mutational spectrum of CDKL5 in early-onset encephalopathies: a study of a large collection of French patients and review of the literature

Inhibition of GSK3β rescues hippocampal development and learning in a mouse model of CDKL5 disorder

Dendritic Spine Instability in a Mouse Model of CDKL5 Disorder Is Rescued by Insulin-like Growth Factor 1

What we know and would like to know about CDKL5 and its involvement in epileptic encephalopathy

P304

9726950

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P31

scholarly article

P356

10.1155/2018/9726950

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P407

English

P478

2018

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P50

Elisabetta Ciani

Laura Gennaccaro

Giorgio Medici

P577

2018-05-27T00:00:00Z

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P698

29977282

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P932

5994305

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