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Endogenous SNAP-25 regulates native voltage-gated calcium channels in glutamatergic neuronsActivity-dependent phosphorylation of Ser187 is required for SNAP-25-negative modulation of neuronal voltage-gated calcium channels.Ectonucleotidase activity and immunosuppression in astrocyte-CD4 T cell bidirectional signaling.Reduced SNAP-25 alters short-term plasticity at developing glutamatergic synapses.The synapsins: multitask modulators of neuronal development.REST-Dependent Presynaptic Homeostasis Induced by Chronic Neuronal Hyperactivity.Lack of IL-1R8 in neurons causes hyperactivation of IL-1 receptor pathway and induces MECP2-dependent synaptic defectsDefects During Mecp2 Null Embryonic Cortex Development Precede the Onset of Overt Neurological Symptoms.Reduced SNAP-25 increases PSD-95 mobility and impairs spine morphogenesis.REST/NRSF-mediated intrinsic homeostasis protects neuronal networks from hyperexcitability.Storage and release of ATP from astrocytes in culture.Calpain activity contributes to the control of SNAP-25 levels in neurons.Analysis of SNAP-25 immunoreactivity in hippocampal inhibitory neurons during development in culture and in situ.SYN1 loss-of-function mutations in autism and partial epilepsy cause impaired synaptic function.Maternal Immune Activation Delays Excitatory-to-Inhibitory Gamma-Aminobutyric Acid Switch in Offspring.Synapsin I deletion reduces neuronal damage and ameliorates clinical progression of experimental autoimmune encephalomyelitis.
P50
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P50
description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Davide Pozzi
@ast
Davide Pozzi
@en
Davide Pozzi
@es
Davide Pozzi
@nl
Davide Pozzi
@sl
type
label
Davide Pozzi
@ast
Davide Pozzi
@en
Davide Pozzi
@es
Davide Pozzi
@nl
Davide Pozzi
@sl
prefLabel
Davide Pozzi
@ast
Davide Pozzi
@en
Davide Pozzi
@es
Davide Pozzi
@nl
Davide Pozzi
@sl
P106
P1153
7003851723
P31
P496
0000-0001-6436-0556