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INaP selective inhibition reverts precocious inter- and motorneurons hyperexcitability in the Sod1-G93R zebrafish ALS modelIdentification of the molecular site of ivabradine binding to HCN4 channelsCyclic dinucleotides bind the C-linker of HCN4 to control channel cAMP responsivenessFunctional comparison of HCN isoforms expressed in ventricular and HEK 293 cellsDeep bradycardia and heart block caused by inducible cardiac-specific knockout of the pacemaker channel gene Hcn4Exercise training reduces resting heart rate via downregulation of the funny channel HCN4Properties of ivabradine-induced block of HCN1 and HCN4 pacemaker channels.The genetic basis for inherited forms of sinoatrial dysfunction and atrioventricular node dysfunctionRegulation of gating and rundown of HCN hyperpolarization-activated channels by exogenous and endogenous PIP2Current-dependent block of rabbit sino-atrial node I(f) channels by ivabradineHeart rate reduction via selective 'funny' channel blockers.Modulation of cyclic nucleotide-regulated HCN channels by PIP(2) and receptors coupled to phospholipase C.Expression of skeletal but not cardiac Na+ channel isoform preserves normal conduction in a depolarized cardiac syncytium.The cardiac pacemaker current.Current understanding of the pathophysiological mechanisms responsible for inappropriate sinus tachycardia: role of the If "funny" current.Cell-specific Dynamic Clamp analysis of the role of funny If current in cardiac pacemaking.Embryonic stem cell-derived CD166+ precursors develop into fully functional sinoatrial-like cells.Wild-type and mutant HCN channels in a tandem biological-electronic cardiac pacemaker.C terminus-mediated control of voltage and cAMP gating of hyperpolarization-activated cyclic nucleotide-gated channels.Recessive loss-of-function mutation in the pacemaker HCN2 channel causing increased neuronal excitability in a patient with idiopathic generalized epilepsy.Erratum: Cyclic dinucleotides bind the C-linker of HCN4 to control channel cAMP responsivenessModulation of rate by autonomic agonists in SAN cells involves changes in diastolic depolarization and the pacemaker current
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description
hulumtuese
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հետազոտող
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Annalisa Bucchi
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Annalisa Bucchi
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Annalisa Bucchi
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Annalisa Bucchi
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Annalisa Bucchi
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Annalisa Bucchi
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Annalisa Bucchi
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Annalisa Bucchi
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Annalisa Bucchi
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Annalisa Bucchi
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Annalisa Bucchi
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Annalisa Bucchi
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Annalisa Bucchi
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Annalisa Bucchi
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Annalisa Bucchi
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Annalisa Bucchi
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P106
P1153
6602103074
P21
P31
P496
0000-0002-5303-4242