about
Phospholipase Cgamma activates Ras on the Golgi apparatus by means of RasGRP1LEF1 in androgen-independent prostate cancer: regulation of androgen receptor expression, prostate cancer growth, and invasionRole of the F-box protein Skp2 in lymphomagenesisCellular response to oncogenic ras involves induction of the Cdk4 and Cdk6 inhibitor p15(INK4b)rgr oncogene: activation by elimination of translational controls and mislocalization.In TCR-stimulated T-cells, N-ras regulates specific genes and signal transduction pathwaysRadiation and chemical activation of ras oncogenes in different mouse strains.ras activation in experimental carcinogenesis.Wild type N-ras displays anti-malignant properties, in part by downregulating decorinp15(INK4b) plays a crucial role in murine lymphoid development and tumorigenesis.Murine leukemia virus sequences are encoded in the murine major histocompatibility complex.Concomitant K- and N-ras gene point mutations in clonal murine lymphomaRas activation in Jurkat T cells following low-grade stimulation of the T-cell receptor is specific to N-Ras and occurs only on the Golgi apparatus.Association of endogenous viral loci with genes encoding murine histocompatibility and lymphocyte differentiation antigens.p53 deficiency provokes urothelial proliferation and synergizes with activated Ha-ras in promoting urothelial tumorigenesis.Regulation of HMGA1 expression by microRNA-296 affects prostate cancer growth and invasionThe differential palmitoylation states of N-Ras and H-Ras determine their distinct Golgi subcompartment localizations.Intraovarian markers of follicular and oocyte maturation.Systemic tumor targeting and killing by Sindbis viral vectors.Complex effects of Ras proto-oncogenes in tumorigenesis.Targeted gene transfer system using a streptavidin-transforming growth factor-alpha chimeric protein.rsc: a novel oncogene with structural and functional homology with the gene family of exchange factors for Ral.NF1 modulates the effects of Ras oncogenes: evidence of other NF1 function besides its GAP activity.Mouse p10, an alternative spliced form of p15INK4b, inhibits cell cycle progression and malignant transformation.Activation of a c-K-ras oncogene by somatic mutation in mouse lymphomas induced by gamma radiation.The Rgr oncogene induces tumorigenesis in transgenic mice.Overexpression of epidermal growth factor receptor in urothelium elicits urothelial hyperplasia and promotes bladder tumor growth.Origin of the tetraspanin uroplakins and their co-evolution with associated proteins: implications for uroplakin structure and function.Using sindbis viral vectors for specific detection and suppression of advanced ovarian cancer in animal models.Isolation of high molecular weight DNA for reliable genotyping of transgenic mice.Hypermethylation of the cell cycle inhibitor p15INK4b 3'-untranslated region interferes with its transcriptional regulation in primary lymphomas.An AC-repeat adjacent to mouse Cdkn2B allows the detection of specific allelic losses in the p15INK4b and p16INK4a tumor suppressor genes.Inactivation of the cyclin-dependent kinase inhibitor p15INK4b by deletion and de novo methylation with independence of p16INK4a alterations in murine primary T-cell lymphomas.Oncogene involvement in tumor regression: H-ras activation in the rabbit keratoacanthoma model.Cloning of a full-length complementary DNA for an Artemia salina glycine-rich protein. Structural relationship with RNA binding proteins.Mouse N-ras genes: organization of the functional locus and of a truncated cDNA-like pseudogene.An overexpressed N-ras proto-oncogene cooperates with N-methylnitrosourea in mouse mammary carcinogenesis.Absence of MDM-2 gene amplification in experimentally induced tumors regardless of p53 status.Primitive neuroectodermal tumors of the brainstem: investigation of seven cases.Phylogenetic relationships among laboratory and wild-origin Mus musculus strains on the basis of genomic DNA RFLPs
P50
Q24307304-34723A9C-EC8A-4F49-B76C-E30CEBD69BEBQ24313620-A3B770C6-1BA1-42A2-8A5C-9B64F3A1FDB9Q24603883-D79A4458-431C-4EB6-B7EA-5803D118E0B9Q28609756-5945789C-144B-4CBE-81BF-71693C476250Q34215956-8FDA8C3F-E455-4CCA-B6E0-707B843D40F5Q34765540-DE450E81-19BD-4813-91BF-09D4DF508E1BQ35032725-0F917BC4-628C-4D89-ACDD-287250AB39CDQ35464212-D0834D7F-3BDA-4CD6-9507-F81FC61FB051Q35539813-67852CFA-3EEB-4013-9892-E80BC557B1F9Q35795388-4497752D-8AA0-47E3-9862-FB1DAE025A1DQ36255856-8EB2AB6E-B964-4BC0-A745-3B56A8DC6871Q36789192-802EA26D-0B42-4585-AE48-B165ADBDDFB8Q37274925-3214E0D9-D337-4E1E-9FF4-28AFABFA012EQ37346114-B41BAEEB-51B0-41A5-B382-60DB6E776AB6Q38345714-C3FEBE95-BE9E-4297-A349-9753E0ACAA26Q38751649-E72EAB45-AC23-4AD0-8285-D5666266E73BQ38962669-017226ED-8575-4C0A-872F-B7248AD8D8D4Q39511360-9FB4B235-D3C5-4F9E-9E84-EA25A5A9ED76Q40612706-907A6E64-D73C-4F56-991F-1A58E406C3B8Q40614418-CF50D081-D702-4CFC-B783-60FA510A8330Q40920854-C46F2A14-0E49-4BAC-B2BD-77DB751C5324Q41120344-582FC4D9-A5D4-49EB-9F3A-AE2BB48E9464Q42795746-CEE3EB10-A2E4-4962-A086-7B2AAE4B35F4Q42815901-C7F75702-2CD1-44A8-BE29-3A6731D50B2FQ42817862-DDFBCD20-37B6-4406-BCC6-64E613AFEE48Q42825949-04C526D4-67A1-4739-93F8-39F0AA63164AQ44068643-06DCC15A-C98A-419A-A0ED-E04EE2CA5A7DQ44266065-FBB80A2F-AB66-42C9-923D-9A51B2EF9263Q45877979-A0704FAD-C122-4BD4-9E4E-85A778ECB49BQ47307938-5997599D-BCFB-4F0D-8B8F-A0B1D21C535DQ47991175-D713B951-BFFD-4F6B-B794-AA820790073FQ48039605-87C4536F-EDD7-4350-B463-246B93F82773Q48052432-1620B9B5-0395-4E7E-AC64-C8D1D4F92DA5Q48230897-A7C2F49D-A7BC-4D42-95AF-8DEFA258E8A8Q48337269-3868AF02-28F2-4C25-AC5F-303CD90763B3Q48342568-BA7B4B73-B870-4ED3-A227-745778B4611CQ53468167-DCF965B3-C5B6-4B16-9390-D074888E5C74Q53474941-B1F81DDA-49CB-45AC-9C45-513105461072Q55456659-E9A0197F-60A0-4E57-B32E-94235F3031E9Q56511642-7BC46888-9A74-44CF-B9DC-761ACCC9D58C
P50
description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
A Pellicer
@ast
A Pellicer
@en
A Pellicer
@es
A Pellicer
@nl
type
label
A Pellicer
@ast
A Pellicer
@en
A Pellicer
@es
A Pellicer
@nl
prefLabel
A Pellicer
@ast
A Pellicer
@en
A Pellicer
@es
A Pellicer
@nl
P106
P31
P496
0000-0002-5062-0692