about
The susceptibility of trypanosomatid pathogens to PI3/mTOR kinase inhibitors affords a new opportunity for drug repurposingThird target of rapamycin complex negatively regulates development of quiescence in Trypanosoma brucei.The AMPKα1 Pathway Positively Regulates the Developmental Transition from Proliferation to Quiescence in Trypanosoma brucei.Establishment of a structure-activity relationship of 1H-imidazo[4,5-c]quinoline-based kinase inhibitor NVP-BEZ235 as a lead for African sleeping sickness.Target of rapamycin (TOR) kinase in Trypanosoma brucei: an extended family.
P50
description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Manuel Saldivia
@ast
Manuel Saldivia
@en
Manuel Saldivia
@es
Manuel Saldivia
@nl
type
label
Manuel Saldivia
@ast
Manuel Saldivia
@en
Manuel Saldivia
@es
Manuel Saldivia
@nl
prefLabel
Manuel Saldivia
@ast
Manuel Saldivia
@en
Manuel Saldivia
@es
Manuel Saldivia
@nl
P106
P21
P31
P496
0000-0001-6204-0460