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Mechanisms of leukemogenesis induced by bovine leukemia virus: prospects for novel anti-retroviral therapies in humanRecent Advances in BLV ResearchModes of Human T Cell Leukemia Virus Type 1 Transmission, Replication and PersistenceBovine Leukemia Virus Small Noncoding RNAs Are Functional Elements That Regulate Replication and Contribute to Oncogenesis In VivoAPOBEC3 Interference during Replication of Viral GenomesVaccination against δ-retroviruses: the bovine leukemia virus paradigmWhole genome sequencing of 51 breast cancers reveals that tumors are devoid of bovine leukemia virus DNAQuantification of HTLV-1 clonality and TCR diversity.Valproate activates bovine leukemia virus gene expression, triggers apoptosis, and induces leukemia/lymphoma regression in vivo.HTLV-1 propels thymic human T cell development in "human immune system" Rag2⁻/⁻ gamma c⁻/⁻ mice.Estimating abundances of retroviral insertion sites from DNA fragment length data.Genome-wide determinants of proviral targeting, clonal abundance and expression in natural HTLV-1 infectionStrongyloidiasis and infective dermatitis alter human T lymphotropic virus-1 clonality in vivoThe host genomic environment of the provirus determines the abundance of HTLV-1-infected T-cell clonesMassive depletion of bovine leukemia virus proviral clones located in genomic transcriptionally active sites during primary infection.Peripheral blood B-cell death compensates for excessive proliferation in lymphoid tissues and maintains homeostasis in bovine leukemia virus-infected sheepSpleen-dependent turnover of CD11b peripheral blood B lymphocytes in bovine leukemia virus-infected sheep.Preventive and therapeutic strategies for bovine leukemia virus: lessons for HTLV.Mutation of a Single Envelope N-Linked Glycosylation Site Enhances the Pathogenicity of Bovine Leukemia Virus.Even attenuated bovine leukemia virus proviruses can be pathogenic in sheepWIP1 deficiency inhibits HTLV-1 Tax oncogenesis: novel therapeutic prospects for treatment of ATL?Cell dynamics and immune response to BLV infection: a unifying model.Histone deacetylase mediated transcriptional activation reduces proviral loads in HTLV-1 associated myelopathy/tropical spastic paraparesis patients.Safety of long-term treatment of HAM/TSP patients with valproic acid.Cis-drivers and trans-drivers of bovine leukemia virus oncogenesis.High-Throughput Mapping and Clonal Quantification of Retroviral Integration Sites.Long-term clinical remission maintained after cessation of zidovudine and interferon-α therapy in chronic adult T-cell leukemia/lymphoma.Quantifying lymphocyte kinetics in vivo using carboxyfluorescein diacetate succinimidyl ester (CFSE).Valproic acid induces apoptosis in chronic lymphocytic leukemia cells through activation of the death receptor pathway and potentiates TRAIL response
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onderzoeker
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հետազոտող
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Nicolas Gillet
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Nicolas Gillet
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Nicolas Gillet
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Nicolas Gillet
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P106
P1153
8646481600
P21
P31
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0000-0002-9410-8738