about
Fluoxetine Prevents Aβ1-42-Induced Toxicity via a Paracrine Signaling Mediated by Transforming-Growth-Factor-β1.Estrogen and Alzheimer's disease: Still an attractive topic despite disappointment from early clinical results.Astrocytes contribute to Aβ-induced blood-brain barrier damage through activation of endothelial MMP9.Estrogen activates matrix metalloproteinases-2 and -9 to increase beta amyloid degradation.Modulation of C6 Glioma Cell Proliferation by Ureido-Calix[8]arenes.Early β-Amyloid-induced Synaptic Dysfunction Is Counteracted by Estrogen in Organotypic Hippocampal Cultures.Integrins mediate beta-amyloid-induced cell-cycle activation and neuronal death.High mobility group box 1 contributes to wound healing induced by inhibition of dipeptidylpeptidase 4 in cultured keratinocytes.Glial metabotropic glutamate receptor-4 increases maturation and survival of oligodendrocytes.Long-term incubation with beta-amyloid peptides impairs endothelium-dependent vasodilatation in isolated rat basilar artery.Glia mediates the neuroprotective action of estradiol on beta-amyloid-induced neuronal death.Targeting group II metabotropic glutamate (mGlu) receptors for the treatment of psychosis associated with Alzheimer's disease: selective activation of mGlu2 receptors amplifies beta-amyloid toxicity in cultured neurons, whereas dual activation of mGEnhanced expression of ERalpha in astrocytes modifies the response of cortical neurons to beta-amyloid toxicity.Dual effect of 17β-estradiol on NMDA-induced neuronal death: involvement of metabotropic glutamate receptor 1.Estrogen receptors and type 1 metabotropic glutamate receptors are interdependent in protecting cortical neurons against β-amyloid toxicity.Chronic Treatment with Fluoxetine Induces Sex-Dependent Analgesic Effects and Modulates HDAC2 and mGlu2 Expression in Female Mice.Shedding of Microvesicles from Microglia Contributes to the Effects Induced by Metabotropic Glutamate Receptor 5 Activation on Neuronal Death.Identification of 5-Methoxyflavone as a Novel DNA Polymerase-Beta Inhibitor and Neuroprotective Agent against Beta-Amyloid Toxicity.The contribution of microglia to early synaptic compensatory responses that precede β-amyloid-induced neuronal death.Early compensatory responses against neuronal injury: A new therapeutic window of opportunity for Alzheimer's Disease?Carnosine Prevents Aβ-Induced Oxidative Stress and Inflammation in Microglial Cells: A Key Role of TGF-β1Distinct effects of pramipexole on the proliferation of adult mouse sub-ventricular zone-derived cells and the appearance of a neuronal phenotypeSIRT1 Mediates Melatonin's Effects on Microglial Activation in Hypoxia: In Vitro and In Vivo EvidenceThe Treatment of Impaired Wound Healing in Diabetes: Looking among Old Drugsβ-amyloid and Oxidative Stress: Perspectives in Drug DevelopmentModulation of Pro-Oxidant and Pro-Inflammatory Activities of M1 Macrophages by the Natural Dipeptide Carnosine
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description
hulumtuese
@sq
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Sara Merlo
@ast
Sara Merlo
@en
Sara Merlo
@es
Sara Merlo
@nl
type
label
Sara Merlo
@ast
Sara Merlo
@en
Sara Merlo
@es
Sara Merlo
@nl
prefLabel
Sara Merlo
@ast
Sara Merlo
@en
Sara Merlo
@es
Sara Merlo
@nl
P106
P1153
16067038700
P21
P31
P496
0000-0002-6558-9800