about
Reproducibility and variability of quantitative magnetic resonance imaging markers in cerebral small vessel disease.The Determinants of Dementia After Stroke (DEDEMAS) Study: protocol and pilot data.Neuroimaging standards for research into small vessel disease and its contribution to ageing and neurodegeneration.ADC histograms from routine DWI for longitudinal studies in cerebral small vessel disease: a field study in CADASIL.Strategic white matter tracts for processing speed deficits in age-related small vessel disease.In vivo high-resolution 7 Tesla MRI shows early and diffuse cortical alterations in CADASIL.Magnetization transfer ratio relates to cognitive impairment in normal elderly.White matter pathology and disconnection in the frontal lobe in cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL).Hippocampal volume is an independent predictor of cognitive performance in CADASIL.Acute infarcts cause focal thinning in remote cortex via degeneration of connecting fiber tractsCerebral Autosomal Dominant Arteriopathy with Subcortical Infarcts and Leukoencephalopathy (CADASIL) as a model of small vessel disease: update on clinical, diagnostic, and management aspects.Consensus statement for diagnosis of subcortical small vessel disease.APOE ɛ2 is associated with white matter hyperintensity volume in CADASILImpact of regional cortical and subcortical changes on processing speed in cerebral small vessel diseaseThe Primacy Effect in Amnestic Mild Cognitive Impairment: Associations with Hippocampal Functional Connectivity.Prevalence of Amyloid Positron Emission Tomographic Positivity in Poststroke Mild Cognitive Impairment.Cerebral Microbleeds and the Risk of Incident Ischemic Stroke in CADASIL (Cerebral Autosomal Dominant Arteriopathy With Subcortical Infarcts and Leukoencephalopathy).Prediction of 3-year clinical course in CADASIL.Neuronal correlates of serial position performance in amnestic mild cognitive impairment.CADASIL mutations enhance spontaneous multimerization of NOTCH3.Cortical Superficial Siderosis in Different Types of Cerebral Small Vessel Disease.Predictors of Clinical Worsening in Cerebral Autosomal Dominant Arteriopathy With Subcortical Infarcts and Leukoencephalopathy: Prospective Cohort Study.Predictors and Clinical Impact of Incident Lacunes in Cerebral Autosomal Dominant Arteriopathy With Subcortical Infarcts and Leukoencephalopathy.Resting-State Connectivity of the Left Frontal Cortex to the Default Mode and Dorsal Attention Network Supports Reserve in Mild Cognitive Impairment.Left Frontal Hub Connectivity during Memory Performance Supports Reserve in Aging and Mild Cognitive Impairment.RNA-Seq Identifies Circulating miR-125a-5p, miR-125b-5p, and miR-143-3p as Potential Biomarkers for Acute Ischemic Stroke.Education modifies the relation of vascular pathology to cognitive function: cognitive reserve in cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy.Identification of a strategic brain network underlying processing speed deficits in vascular cognitive impairment.Carotid atherosclerotic markers in CADASIL.Strategic role of frontal white matter tracts in vascular cognitive impairment: a voxel-based lesion-symptom mapping study in CADASIL.Left frontal cortex connectivity underlies cognitive reserve in prodromal Alzheimer disease.Regulation of cholesterol and sphingomyelin metabolism by amyloid-beta and presenilin.Microstructure of Strategic White Matter Tracts and Cognition in Memory Clinic Patients with Vascular Brain Injury.Remote changes after ischaemic infarcts: a distant target for therapy?Incident lacunes preferentially localize to the edge of white matter hyperintensities: insights into the pathophysiology of cerebral small vessel disease.Prevalence and characteristics of migraine in CADASIL.Lower Magnetization Transfer Ratio in the Forceps Minor Is Associated with Poorer Gait Velocity in Older Adults.NIHSS scores in ischemic small vessel disease: a study in CADASIL.Incident subcortical infarcts induce focal thinning in connected cortical regions.Cysteine-sparing CADASIL mutations in NOTCH3 show proaggregatory properties in vitro.
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P50
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wetenschapper
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հետազոտող
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Marco Duering
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Marco Duering
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Marco Duering
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Marco Duering
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Marco Duering
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Marco Duering
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Marco Duering
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Marco Duering
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Marco Duering
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Marco Duering
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Marco Duering
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Marco Duering
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0000-0003-2302-3136