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Collybistin splice variants differentially interact with gephyrin and Cdc42 to regulate gephyrin clustering at GABAergic synapses.Mutations in NONO lead to syndromic intellectual disability and inhibitory synaptic defectsSelective regulation of NR2B by protein phosphatase-1 for the control of the NMDA receptor in neuroprotection.Stability and function of JC virus large T antigen and T' proteins are altered by mutation of their phosphorylated threonine 125 residues.Regulation of GABAergic synapse formation and plasticity by GSK3beta-dependent phosphorylation of gephyrin.Activity-dependent inhibitory synapse remodeling through gephyrin phosphorylation.Extracellular signal-regulated kinase and glycogen synthase kinase 3β regulate gephyrin postsynaptic aggregation and GABAergic synaptic function in a calpain-dependent mechanism.Mitochondrial reactive oxygen species regulate the strength of inhibitory GABA-mediated synaptic transmissionSeveral posttranslational modifications act in concert to regulate gephyrin scaffolding and GABAergic transmission.GABA(A) receptors, gephyrin and homeostatic synaptic plasticity.Molecular and functional heterogeneity of GABAergic synapses.Gephyrin: a master regulator of neuronal function?Nanoscale Molecular Reorganization of the Inhibitory Postsynaptic Density Is a Determinant of GABAergic Synaptic Potentiation.JC virus T'135, T'136 and T'165 proteins interact with cellular p107 and p130 in vivo and influence viral transformation potential.Partial inactivation of GABAA receptors containing the α5 subunit affects the development of adult-born dentate gyrus granule cells.Postsynaptic gephyrin clustering controls the development of adult-born granule cells in the olfactory bulb.Basic presynaptic functions in hippocampal neurons are not affected by acute or chronic lithium treatment.RhoGEF9 splice isoforms influence neuronal maturation and synapse formation downstream of α2 GABAA receptors.Emerging Mechanisms Underlying Dynamics of GABAergic Synapses.Estradiol modulates the efficacy of synaptic inhibition by decreasing the dwell time of GABAA receptors at inhibitory synapses.Activity-Dependent Inhibitory Synapse Scaling Is Determined by Gephyrin Phosphorylation and Subsequent Regulation of GABAA Receptor Diffusion.Neuronal Dystroglycan Is Necessary for Formation and Maintenance of Functional CCK-Positive Basket Cell Terminals on Pyramidal Cells.Distinct mechanisms regulate GABAA receptor and gephyrin clustering at perisomatic and axo-axonic synapses on CA1 pyramidal cells.The forebrain synaptic transcriptome is organized by clocks but its proteome is driven by sleepSleep-wake cycles drive daily dynamics of synaptic phosphorylationThreat Memory Reminder Under Matrix Metalloproteinase 9 Inhibitor Doxycycline Globally Reduces Subsequent Memory Plasticity
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description
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Shiva K Tyagarajan
@ast
Shiva K Tyagarajan
@en
Shiva K Tyagarajan
@es
Shiva K Tyagarajan
@nl
type
label
Shiva K Tyagarajan
@ast
Shiva K Tyagarajan
@en
Shiva K Tyagarajan
@es
Shiva K Tyagarajan
@nl
prefLabel
Shiva K Tyagarajan
@ast
Shiva K Tyagarajan
@en
Shiva K Tyagarajan
@es
Shiva K Tyagarajan
@nl
P106
P1153
54950947800
P31
P496
0000-0003-0074-1805