Understanding Podocyte Biology to Develop Novel Kidney Therapeutics

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Understanding Podocyte Biology to Develop Novel Kidney Therapeutics

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wetenschappelijk artikel

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наукова стаття, опублікована у 2018

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Understanding Podocyte Biology to Develop Novel Kidney Therapeutics

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Understanding Podocyte Biology to Develop Novel Kidney Therapeutics

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Understanding Podocyte Biology to Develop Novel Kidney Therapeutics

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Understanding Podocyte Biology to Develop Novel Kidney Therapeutics

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Understanding Podocyte Biology to Develop Novel Kidney Therapeutics

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Understanding Podocyte Biology to Develop Novel Kidney Therapeutics

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P1476

Understanding Podocyte Biology to Develop Novel Kidney Therapeutics

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P2093

Jaakko Patrakka

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Mark A Lal

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P2860

COQ6 mutations in human patients produce nephrotic syndrome with sensorineural deafness.

Molecular mechanisms of diabetic kidney disease

Modelling kidney disease with CRISPR-mutant kidney organoids derived from human pluripotent epiblast spheroids

Podocyte developmental defects caused by adriamycin in zebrafish embryos and larvae: a novel model of glomerular damage

Mutations in ACTN4, encoding alpha-actinin-4, cause familial focal segmental glomerulosclerosis

Establishment of Nephrin Reporter Mice and Use for Chemical Screening

TRPC6 is a glomerular slit diaphragm-associated channel required for normal renal function

Inducible podocyte injury and proteinuria in transgenic zebrafish.

Rituximab targets podocytes in recurrent focal segmental glomerulosclerosis.

Inhibition of the TRPC5 ion channel protects the kidney filter

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409

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scholarly article

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10.3389/FENDO.2018.00409

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English

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2018-01-01T00:00:00Z

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30083135

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6065143

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