about
Mitochondria-targeted dodecyltriphenylphosphonium (C12TPP) combats high-fat-diet-induced obesity in miceActivation of AMPKα2 is not crucial for mitochondrial uncoupling-induced metabolic effects but required to maintain skeletal muscle integrityTargeted mitochondrial uncoupling beyond UCP1 - The fine line between death and metabolic health.Skeletal muscle mitochondrial uncoupling drives endocrine cross-talk through the induction of FGF21 as a myokine.Muscle mitochondrial stress adaptation operates independently of endogenous FGF21 action.Skeletal muscle uncoupling-induced longevity in mice is linked to increased substrate metabolism and induction of the endogenous antioxidant defense system.Regulation of myokine expression: Role of exercise and cellular stress.Long-Term Cold Adaptation Does Not Require FGF21 or UCP1.Partial involvement of Nrf2 in skeletal muscle mitohormesis as an adaptive response to mitochondrial uncoupling.Muscle mitohormesis promotes cellular survival via serine/glycine pathway flux.ELEVATED SERUM GROWTH DIFFERENTIATION FACTOR 15 LEVELS IN GERIATRIC PATIENTS – SARCOPENIA AND PHYSICAL PARAMETERSHIGHER SERUM LEVELS OF FIBROBLAST GROWTH FACTOR 21 IN GERIATRIC PATIENTS WITH CACHEXIAAuthor Correction: Partial involvement of Nrf2 in skeletal muscle mitohormesis as an adaptive response to mitochondrial uncouplingMuscle-derived GDF15 drives diurnal anorexia and systemic metabolic remodeling during mitochondrial stressMitochondrial uncoupling and longevity - A role for mitokines?Role of GDF15 in active lifestyle induced metabolic adaptations and acute exercise response in miceHigher serum levels of fibroblast growth factor 21 in old patients with cachexia
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description
researcher
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wetenschapper
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հետազոտող
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name
M Ost
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M Ost
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M Ost
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M Ost
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M Ost
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M Ost
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M Ost
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M Ost
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M Ost
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M Ost
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M Ost
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M Ost
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P31
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0000-0002-1811-2049