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Myocyte Enhancer Factors 2A and 2C Induce Dilated Cardiomyopathy in Transgenic Mice

Myocyte Enhancer Factors 2A and 2C Induce Dilated Cardiomyopathy in Transgenic Mice

http://www.wikidata.org/entity/Q57396513

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Genome-wide association of echocardiographic dimensions, brachial artery endothelial function and treadmill exercise responses in the Framingham Heart Study Kruppel-like factor 15 is a regulator of cardiomyocyte hypertrophy.Histone deacetylase signaling in cardioprotection MicroRNA-1 negatively regulates expression of the hypertrophy-associated calmodulin and Mef2a genes Modulation of adverse cardiac remodeling by STARS, a mediator of MEF2 signaling and SRF activity Troponin I-interacting protein kinase: a novel cardiac-specific kinase, emerging as a molecular target for the treatment of cardiac disease Calreticulin induces dilated cardiomyopathy CIP, a cardiac Isl1-interacting protein, represses cardiomyocyte hypertrophy Myocyte enhancer factor 2c, an osteoblast transcription factor identified by dimethyl sulfoxide (DMSO)-enhanced mineralization Regulation of lymphoid versus myeloid fate 'choice' by the transcription factor Mef2c Ablation of the cardiac-specific gene leucine-rich repeat containing 10 (Lrrc10) results in dilated cardiomyopathy Polycystin-dependent fluid flow sensing targets histone deacetylase 5 to prevent the development of renal cysts Modulation of chromatin position and gene expression by HDAC4 interaction with nucleoporins Myomaxin is a novel transcriptional target of MEF2A that encodes a Xin-related alpha-actinin-interacting protein The positive transcription elongation factor b is an essential cofactor for the activation of transcription by myocyte enhancer factor 2 Construction of microRNA and transcription factor regulatory network based on gene expression data in cardiomyopathy Differentiation of human adipose tissue SVF cells into cardiomyocytes.MEF2C silencing attenuates load-induced left ventricular hypertrophy by modulating mTOR/S6K pathway in mice Novel therapeutic effects of sesamin on diabetes-induced cardiac dysfunction.Steroid receptor coactivator-2 is a dual regulator of cardiac transcription factor function.Heart Failure and MEF2 Transcriptome Dynamics in Response to β-Blockers.Regulation of cardiomyocyte Glut4 expression by ZAC1 Mitsugumin 29 regulates t-tubule architecture in the failing heart.Severe mental retardation, seizures, and hypotonia due to deletions of MEF2C.Left ventricular global transcriptional profiling in human end-stage dilated cardiomyopathy.Genome wide response to dietary tetradecylthioacetic acid supplementation in the heart of Atlantic Salmon (Salmo salar L).A novel microdeletion syndrome involving 5q14.3-q15: clinical and molecular cytogenetic characterization of three patients Loss of Frataxin induces iron toxicity, sphingolipid synthesis, and Pdk1/Mef2 activation, leading to neurodegeneration.Extracellular signal-regulated kinases 1 and 2 regulate the balance between eccentric and concentric cardiac growth Loss of Frataxin activates the iron/sphingolipid/PDK1/Mef2 pathway in mammals Genetic loci for retinal arteriolar microcirculation.MEF2 transcription factors regulate distinct gene programs in mammalian skeletal muscle differentiation Evidence for coregulation of myocardial gene expression by MEF2 and NFAT in human heart failure Anabolic androgenic steroids and intracellular calcium signaling: a mini review on mechanisms and physiological implications.Re-employment of developmental transcription factors in adult heart disease Bnip3 Binds and Activates p300: Possible Role in Cardiac Transcription and Myocyte Morphology.Protein kinase C (PKC)ζ-mediated Gαq stimulation of ERK5 protein pathway in cardiomyocytes and cardiac fibroblasts.Transcription factor MEF2C suppresses endothelial cell inflammation via regulation of NF-κB and KLF2 MEF2C ablation in endothelial cells reduces retinal vessel loss and suppresses pathologic retinal neovascularization in oxygen-induced retinopathy The MEF2D transcription factor mediates stress-dependent cardiac remodeling in mice.

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Myocyte Enhancer Factors 2A and 2C Induce Dilated Cardiomyopathy in Transgenic Mice

http://www.wikidata.org/entity/Q57396513

description

article publié dans la revue scientifique Journal of Biological Chemistry

@fr

im Februar 2006 veröffentlichter wissenschaftlicher Artikel

@de

scientific article published in Journal of Biological Chemistry

@en

wetenschappelijk artikel

@nl

наукова стаття, опублікована в лютому 2006

@uk

name

Myocyte Enhancer Factors 2A and 2C Induce Dilated Cardiomyopathy in Transgenic Mice

@en

Myocyte Enhancer Factors 2A and 2C Induce Dilated Cardiomyopathy in Transgenic Mice

@nl

type

label

Myocyte Enhancer Factors 2A and 2C Induce Dilated Cardiomyopathy in Transgenic Mice

@en

Myocyte Enhancer Factors 2A and 2C Induce Dilated Cardiomyopathy in Transgenic Mice

@nl

prefLabel

Myocyte Enhancer Factors 2A and 2C Induce Dilated Cardiomyopathy in Transgenic Mice

@en

Myocyte Enhancer Factors 2A and 2C Induce Dilated Cardiomyopathy in Transgenic Mice

@nl

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Journal of Biological Chemistry

P1476

Myocyte enhancer factors 2A and 2C induce dilated cardiomyopathy in transgenic mice

@en

P2093

Ilona Bodi

http://www.w3.org/2001/XMLSchema#string

Jian Xu

http://www.w3.org/2001/XMLSchema#string

Nanling L Gong

http://www.w3.org/2001/XMLSchema#string

Peter H Backx

http://www.w3.org/2001/XMLSchema#string

P2860

Regulation of the MEF2 family of transcription factors by p38

Targeting of p38 mitogen-activated protein kinases to MEF2 transcription factors

Regulation of skeletal myogenesis by association of the MEF2 transcription factor with class II histone deacetylases

Signal-dependent nuclear export of a histone deacetylase regulates muscle differentiation

BMK1/ERK5 regulates serum-induced early gene expression through transcription factor MEF2C

MEF-2 function is modified by a novel co-repressor, MITR

HDAC4 deacetylase associates with and represses the MEF2 transcription factor

Activated MEK5 induces serial assembly of sarcomeres and eccentric cardiac hypertrophy.

The DnaJ-related factor Mrj interacts with nuclear factor of activated T cells c3 and mediates transcriptional repression through class II histone deacetylase recruitment

Identification of a signal-responsive nuclear export sequence in class II histone deacetylases

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9152-9162

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scholarly article

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10.1074/JBC.M510217200

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14

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281

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Jeffery D. Molkentin

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2006-02-09T00:00:00Z

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16469744

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